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C E Shaw

Showing results (41-50 of 76) with videos related to

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The Journal of Clinical Endocrinology and Metabolism|November 1, 1991
3 beta-Hydroxysteroid dehydrogenase activity in glandular and extraglandular human fetal tissuesL Milewich, C E Shaw, K M Doody, et al.
The Journal of Steroid Biochemistry and Molecular Biology|June 1, 1993
3 beta-hydroxysteroid dehydrogenase activity in tissues of the human fetus determined with 5 alpha-androstane-3 beta,17 beta-diol and dehydroepiandrosterone as substratesL Milewich, C E Shaw, J I Mason, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|September 24, 2010
An estimate of amyotrophic lateral sclerosis heritability using twin dataA Al-Chalabi, F Fang, M F Hanby, et al.
American Journal of Physiology. Heart and Circulatory Physiology|March 15, 2001
Endothelial vasodilator production by uterine and systemic arteries. VII. Estrogen and progesterone effects on eNOSH L Rupnow, T M Phernetton, C E Shaw, et al.
Journal of Neurology|March 3, 2009
SOD1 and cognitive dysfunction in familial amyotrophic lateral sclerosisP Wicks, S Abrahams, B Papps, et al.
The American Journal of Physiology|November 14, 1998
Endothelial vasodilator production by uterine and systemic arteries. III. Ovarian and estrogen effects on NO synthaseK E Vagnoni, C E Shaw, T M Phernetton, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|February 14, 2008
CHMP2B mutations are not a common cause of familial or sporadic amyotrophic lateral sclerosisI P Blair, C Vance, J C Durnall, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|July 15, 2000
Phenotypic differences between African and white patients with motor neuron disease: a case-control studyB Tomik, A Nicotra, C M Ellis, et al.
Neurology|December 31, 1997
Familial amyotrophic lateral sclerosis. Molecular pathology of a patient with a SOD1 mutationC E Shaw, Z E Enayat, J F Powell, et al.
Annals of Neurology|March 20, 1998
Mutations in all five exons of SOD-1 may cause ALSC E Shaw, Z E Enayat, B A Chioza, et al.
Pageof 8

Showing results (41-50 of 76) with videos related to

Sort By:
Pageof 8
The Journal of Clinical Endocrinology and Metabolism|November 1, 1991
3 beta-Hydroxysteroid dehydrogenase activity in glandular and extraglandular human fetal tissuesL Milewich, C E Shaw, K M Doody, et al.
The Journal of Steroid Biochemistry and Molecular Biology|June 1, 1993
3 beta-hydroxysteroid dehydrogenase activity in tissues of the human fetus determined with 5 alpha-androstane-3 beta,17 beta-diol and dehydroepiandrosterone as substratesL Milewich, C E Shaw, J I Mason, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|September 24, 2010
An estimate of amyotrophic lateral sclerosis heritability using twin dataA Al-Chalabi, F Fang, M F Hanby, et al.
American Journal of Physiology. Heart and Circulatory Physiology|March 15, 2001
Endothelial vasodilator production by uterine and systemic arteries. VII. Estrogen and progesterone effects on eNOSH L Rupnow, T M Phernetton, C E Shaw, et al.
Journal of Neurology|March 3, 2009
SOD1 and cognitive dysfunction in familial amyotrophic lateral sclerosisP Wicks, S Abrahams, B Papps, et al.
The American Journal of Physiology|November 14, 1998
Endothelial vasodilator production by uterine and systemic arteries. III. Ovarian and estrogen effects on NO synthaseK E Vagnoni, C E Shaw, T M Phernetton, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|February 14, 2008
CHMP2B mutations are not a common cause of familial or sporadic amyotrophic lateral sclerosisI P Blair, C Vance, J C Durnall, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|July 15, 2000
Phenotypic differences between African and white patients with motor neuron disease: a case-control studyB Tomik, A Nicotra, C M Ellis, et al.
Neurology|December 31, 1997
Familial amyotrophic lateral sclerosis. Molecular pathology of a patient with a SOD1 mutationC E Shaw, Z E Enayat, J F Powell, et al.
Annals of Neurology|March 20, 1998
Mutations in all five exons of SOD-1 may cause ALSC E Shaw, Z E Enayat, B A Chioza, et al.
Pageof 8