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Molecular Psychiatry|May 13, 1998
Antipsychotic drugs which elicit little or no parkinsonism bind more loosely than dopamine to brain D2 receptors, yet occupy high levels of these receptorsP Seeman, T TallericoThe American Journal of Psychiatry|June 9, 1999
Rapid release of antipsychotic drugs from dopamine D2 receptors: an explanation for low receptor occupancy and early clinical relapse upon withdrawal of clozapine or quetiapineP Seeman, T TallericoNaunyn-Schmiedeberg'S Archives of Pharmacology|January 1, 1986
[3H]-domperidone labels only a single population of receptors which convert from high to low affinity for dopamine in rat brainD Grigoriadis, P SeemanScience (New York, N.Y.)|June 20, 1975
Antipsychotic drugs: direct correlation between clinical potency and presynaptic action on dopamine neuronsP Seeman, T LeeProceedings of the National Academy of Sciences of the United States of America|May 1, 1978
Selective labeling of alpha-adrenergic receptors in caudate nucleus by [3H] dihydroergocryptine in the presence of spiperone-blocked dopamine receptorsM Titeler, P SeemanDrug and Alcohol Dependence|January 1, 1979
Mechanisms of ethanol tolerance at a cholinergic nerve terminalM Curran, P SeemanThe American Journal of Psychiatry|March 7, 2001
Does fast dissociation from the dopamine d(2) receptor explain the action of atypical antipsychotics?: A new hypothesisS Kapur, P SeemanEuropean Journal of Pharmacology|April 15, 1979
Presynaptic subsensitivity as a possible basis for sensitization by long-term dopamine mimeticsP Muller, P SeemanNeurochemistry International|May 27, 2010
Dopamine receptors in brain and peripheryP Seeman, D GrigoriadisJournal of Psychiatry & Neuroscience : JPN|March 31, 2000
Antipsychotic agents differ in how fast they come off the dopamine D2 receptors. Implications for atypical antipsychotic actionS Kapur, P SeemanPageof 23