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Cell Metabolism|January 10, 2006
The mitochondria and insulin release: Nnt just a passing relationshipM S Remedi, C G Nichols, J C KosterDiabetes, Obesity & Metabolism|October 9, 2007
beta-cell hyperexcitability: from hyperinsulinism to diabetesC G Nichols, J C Koster, M S RemediDiabetes, Obesity & Metabolism|August 30, 2012
The diabetic β-cell: hyperstimulated vs. hyperexcitedC G Nichols, M S RemediAmerican Journal of Physiology. Endocrinology and Metabolism|August 10, 2002
Diabetes and insulin secretion: whither KATP?C G Nichols, J C KosterProceedings of the National Academy of Sciences of the United States of America|December 18, 2002
Hyperinsulinism induced by targeted suppression of beta cell KATP channelsJ C Koster, M S Remedi, T P Flagg, et al.Diabetologia|August 23, 2006
Hyperinsulinism in mice with heterozygous loss of K(ATP) channelsM S Remedi, J V Rocheleau, A Tong, et al.The Journal of General Physiology|August 6, 1999
Sulfonylurea and K(+)-channel opener sensitivity of K(ATP) channels. Functional coupling of Kir6.2 and SUR1 subunitsJ C Koster, Q Sha, C G NicholsThe Journal of Physiology|February 2, 1999
ATP inhibition of KATP channels: control of nucleotide sensitivity by the N-terminal domain of the Kir6.2 subunitJ C Koster, Q Sha, S Shyng, et al.Biophysical Journal|April 17, 1998
Assembly of ROMK1 (Kir 1.1a) inward rectifier K+ channel subunits involves multiple interaction sitesJ C Koster, K A Bentle, C G Nichols, et al.American Journal of Physiology. Heart and Circulatory Physiology|July 19, 2002
Contractility and ischemic response of hearts from transgenic mice with altered sarcolemmal K(ATP) channelsR Rajashree, J C Koster, K P Markova, et al.Pageof 10