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Diabetes, Obesity & Metabolism|August 30, 2012
The diabetic β-cell: hyperstimulated vs. hyperexcitedC G Nichols, M S RemediCell Metabolism|January 10, 2006
The mitochondria and insulin release: Nnt just a passing relationshipM S Remedi, C G Nichols, J C KosterDiabetes, Obesity & Metabolism|October 9, 2007
beta-cell hyperexcitability: from hyperinsulinism to diabetesC G Nichols, J C Koster, M S RemediDiabetologia|January 29, 2011
Defects in beta cell Ca²+ signalling, glucose metabolism and insulin secretion in a murine model of K(ATP) channel-induced neonatal diabetes mellitusR K P Benninger, M S Remedi, W S Head, et al.Proceedings of the National Academy of Sciences of the United States of America|December 18, 2002
Hyperinsulinism induced by targeted suppression of beta cell KATP channelsJ C Koster, M S Remedi, T P Flagg, et al.Diabetes, Obesity & Metabolism|September 13, 2016
Pancreatic β-cell identity in diabetesM S Remedi, C EmfingerDiabetologia|August 23, 2006
Hyperinsulinism in mice with heterozygous loss of K(ATP) channelsM S Remedi, J V Rocheleau, A Tong, et al.The Journal of Physiology|April 1, 1985
The influence of 'diastolic' length on the contractility of isolated cat papillary muscleC G NicholsThe Journal of General Physiology|January 10, 1998
Octameric stoichiometry of the KATP channel complexS Shyng, C G NicholsAmerican Journal of Physiology. Endocrinology and Metabolism|August 10, 2002
Diabetes and insulin secretion: whither KATP?C G Nichols, J C KosterPageof 10