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Natural Immunity and Cell Growth Regulation|January 1, 1989
Regulation of human natural cytotoxicity by IgG. III. Interaction between negative regulation by monomeric IgG and positive regulation by interferons of different typesA Sulica, M Gherman, M Manciulea, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 1, 1982
Inhibition of human natural killer cell activity by cytophilic immunoglobulin GA Sulica, M Gherman, C Galatiuc, et al.
Cellular Immunology|July 1, 1995
Natural killer (NK) activity in human responders and nonresponders to stimulation by anti-CD16 antibodiesC Galatiuc, M Gherman, D Metes, et al.
Natural Immunity and Cell Growth Regulation|January 1, 1989
Effect of interleukin-2 on the monomeric IgG-induced inhibition of human natural killer cell activityM Gherman, R B Herberman, A Sulica
European Journal of Immunology|December 1, 1979
IgG-binding sites on macrophage cell membrane. II. Mobility of Fc receptors induced by the interaction with their corresponding IgG ligandsA Sulica, M Gherman, C Medeşan, et al.
European Journal of Immunology|December 1, 1979
IgG-binding sites on macrophage cell membrane. I. Identification of two distinct Fc receptors on mouse peritoneal macrophagesA Sulica, M Gherman, C Medeşan, et al.
Scandinavian Journal of Immunology|January 1, 1976
Arming of lymphoid cells by IgG antibodies treated with protein A from Staphylococcus aureusA Sulica, M Laky, M Gherman, et al.
Clinical Immunology and Immunopathology|June 1, 1991
Expression of Fc mu receptors on human natural killer cellsL Pricop, C Galatiuc, M Manciulea, et al.
European Journal of Immunology|June 1, 1996
Divergent effects of Fc gamma RIIIA ligands on the functional activities of human natural killer cells in vitroA Sulica, D Metes, M Gherman, et al.
Cellular Immunology|April 1, 1993
Regulation of human natural cytotoxicity by IgG. IV. Association between binding of monomeric IgG to the Fc receptors on large granular lymphocytes and inhibition of natural killer (NK) cell activityA Sulica, C Galatiuc, M Manciulea, et al.
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