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Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|July 31, 1998
Tissue architecture: the ultimate regulator of epithelial function?C Hagios, A Lochter, M J Bissell
APMIS : Acta Pathologica, Microbiologica, Et Immunologica Scandinavica|April 6, 1999
An odyssey from breast to bone: multi-step control of mammary metastases and osteolysis by matrix metalloproteinasesA Lochter, M J Bissell
Seminars in Cancer Biology|June 1, 1995
Involvement of extracellular matrix constituents in breast cancerA Lochter, M J Bissell
Seminars in Cancer Biology|April 27, 2001
Tumors are unique organs defined by abnormal signaling and contextD Radisky, C Hagios, M J Bissell
Matrix Biology : Journal of the International Society for Matrix Biology|December 22, 1999
Transcriptional regulation of stromelysin-1 gene expression is altered during progression of mouse mammary epithelial cells from functionally normal to malignantA Lochter, Z Werb, M J Bissell
Molecular Biology of the Cell|February 9, 1999
alpha1 and alpha2 integrins mediate invasive activity of mouse mammary carcinoma cells through regulation of stromelysin-1 expressionA Lochter, M Navre, Z Werb, et al.
Annals of the New York Academy of Sciences|January 26, 1999
The significance of matrix metalloproteinases during early stages of tumor progressionA Lochter, M D Sternlicht, Z Werb, et al.
Molecular Biology of the Cell|September 3, 1999
Division of labor among the alpha6beta4 integrin, beta1 integrins, and an E3 laminin receptor to signal morphogenesis and beta-casein expression in mammary epithelial cellsJ Muschler, A Lochter, C D Roskelley, et al.
Development (Cambridge, England)|November 2, 2001
The interplay of matrix metalloproteinases, morphogens and growth factors is necessary for branching of mammary epithelial cellsM Simian, Y Hirai, M Navre, et al.
The Journal of Cell Biology|February 7, 1998
Matrix metalloproteinase stromelysin-1 triggers a cascade of molecular alterations that leads to stable epithelial-to-mesenchymal conversion and a premalignant phenotype in mammary epithelial cellsA Lochter, S Galosy, J Muschler, et al.
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