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Molecular and Cellular Biology|March 1, 1992
Progression of colorectal cancer is associated with multiple tumor suppressor gene defects but inhibition of tumorigenicity is accomplished by correction of any single defect via chromosome transferM C Goyette, K Cho, C L Fasching, et al.
Oncogene|May 15, 2007
Targeting the Raf-MEK-ERK mitogen-activated protein kinase cascade for the treatment of cancerP J Roberts, C J Der
Breast Cancer Research and Treatment|July 1, 1995
Aberrant function of the Ras signal transduction pathway in human breast cancerG J Clark, C J Der
Current Opinion in Cell Biology|December 1, 1992
Protein prenylation: more than just glue?A D Cox, C J Der
Oncogene|August 9, 2001
Link of a new type of apoptosis-inducing gene ASY/Nogo-B to human cancerQ Li, B Qi, K Oka, et al.
Genes, Chromosomes & Cancer|March 1, 1997
A 3p21.3 region is preferentially eliminated from human chromosome 3/mouse microcell hybrids during tumor growth in SCID miceI Kholodnyuk, M Kost-Alimova, V Kashuba, et al.
Current Opinion in Cell Biology|March 14, 2000
The Ras branch of small GTPases: Ras family members don't fall far from the treeG W Reuther, C J Der
Methods in Enzymology|January 1, 1994
Biological assays for cellular transformationA D Cox, C J Der
Critical Reviews in Oncogenesis|January 1, 1992
The ras/cholesterol connection: implications for ras oncogenicityA D Cox, C J Der
Cancer Cells (Cold Spring Harbor, N.Y. : 1989)|September 1, 1991
Isoprenoid modification and plasma membrane association: critical factors for ras oncogenicityC J Der, A D Cox
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