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Oncogene|June 27, 2006
Identification of a tumor suppressive critical region mapping to 3p14.2 in esophageal squamous cell carcinoma and studies of a candidate tumor suppressor gene, ADAMTS9P H Y Lo, A C C Leung, C Y C Kwok, et al.
Genomics|July 1, 1996
The construction of a yeast artificial chromosome (YAC) contig in the vicinity of the Usher syndrome type IIa (USH2A) gene in 1q41J Sumegi, J Y Wang, D K Zhen, et al.
The Journal of Biological Chemistry|June 27, 1998
Transforming potential of Dbl family proteins correlates with transcription from the cyclin D1 promoter but not with activation of Jun NH2-terminal kinase, p38/Mpk2, serum response factor, or c-JunJ K Westwick, R J Lee, Q T Lambert, et al.
Oncogene|May 13, 1999
TC21 and Ras share indistinguishable transforming and differentiating activitiesS M Graham, S M Oldham, C B Martin, et al.
Oncogene|August 22, 2000
G2A is an oncogenic G protein-coupled receptorI E Zohn, M Klinger, X Karp, et al.
Oncogene|May 22, 2001
The thrombin receptor, PAR-1, causes transformation by activation of Rho-mediated signaling pathwaysC B Martin, G M Mahon, M B Klinger, et al.
The Prostate|January 1, 1988
Expression of ras proto-oncogenes in the Dunning R3327 rat prostatic adenocarcinoma systemD B Cooke, V E Quarmby, P Petrusz, et al.
Gastroenterology|August 28, 1999
Pharmacological inhibition of Ras-transformed epithelial cell growth is linked to down-regulation of epidermal growth factor-related peptidesN Sizemore, A D Cox, J A Barnard, et al.
The Journal of Biological Chemistry|April 1, 2000
Vav2 is an activator of Cdc42, Rac1, and RhoAK Abe, K L Rossman, B Liu, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 28, 1995
Biological and structural characterization of a Ras transforming mutation at the phenylalanine-156 residue, which is conserved in all members of the Ras superfamilyL A Quilliam, S Zhong, K M Rabun, et al.
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