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Proceedings of the National Academy of Sciences of the United States of America
|
June 21, 1994
Oncogenic Ras activates c-Jun via a separate pathway from the activation of extracellular signal-regulated kinases
J K Westwick, A D Cox, C J Der, et al.
Oncogene
|
October 20, 2009
Differential requirement of CAAX-mediated posttranslational processing for Rheb localization and signaling
A B Hanker, N Mitin, R S Wilder, et al.
Journal of Immunology (Baltimore, Md. : 1950)
|
September 1, 1991
Rap1A is a substrate for cyclic AMP-dependent protein kinase in human neutrophils
L A Quilliam, H Mueller, B P Bohl, et al.
The Journal of Biological Chemistry
|
June 27, 1998
Transforming potential of Dbl family proteins correlates with transcription from the cyclin D1 promoter but not with activation of Jun NH2-terminal kinase, p38/Mpk2, serum response factor, or c-Jun
J K Westwick, R J Lee, Q T Lambert, et al.
Oncogene
|
May 13, 1999
TC21 and Ras share indistinguishable transforming and differentiating activities
S M Graham, S M Oldham, C B Martin, et al.
Oncogene
|
August 22, 2000
G2A is an oncogenic G protein-coupled receptor
I E Zohn, M Klinger, X Karp, et al.
Oncogene
|
May 22, 2001
The thrombin receptor, PAR-1, causes transformation by activation of Rho-mediated signaling pathways
C B Martin, G M Mahon, M B Klinger, et al.
The Prostate
|
January 1, 1988
Expression of ras proto-oncogenes in the Dunning R3327 rat prostatic adenocarcinoma system
D B Cooke, V E Quarmby, P Petrusz, et al.
Gastroenterology
|
August 28, 1999
Pharmacological inhibition of Ras-transformed epithelial cell growth is linked to down-regulation of epidermal growth factor-related peptides
N Sizemore, A D Cox, J A Barnard, et al.
The Journal of Biological Chemistry
|
April 1, 2000
Vav2 is an activator of Cdc42, Rac1, and RhoA
K Abe, K L Rossman, B Liu, et al.
Page
of 18
Search research articles
Search
Showing results (121-130 of 172) with videos related to
Sort By:
Page
of 18
Proceedings of the National Academy of Sciences of the United States of America
|
June 21, 1994
Oncogenic Ras activates c-Jun via a separate pathway from the activation of extracellular signal-regulated kinases
J K Westwick, A D Cox, C J Der, et al.
Oncogene
|
October 20, 2009
Differential requirement of CAAX-mediated posttranslational processing for Rheb localization and signaling
A B Hanker, N Mitin, R S Wilder, et al.
Journal of Immunology (Baltimore, Md. : 1950)
|
September 1, 1991
Rap1A is a substrate for cyclic AMP-dependent protein kinase in human neutrophils
L A Quilliam, H Mueller, B P Bohl, et al.
The Journal of Biological Chemistry
|
June 27, 1998
Transforming potential of Dbl family proteins correlates with transcription from the cyclin D1 promoter but not with activation of Jun NH2-terminal kinase, p38/Mpk2, serum response factor, or c-Jun
J K Westwick, R J Lee, Q T Lambert, et al.
Oncogene
|
May 13, 1999
TC21 and Ras share indistinguishable transforming and differentiating activities
S M Graham, S M Oldham, C B Martin, et al.
Oncogene
|
August 22, 2000
G2A is an oncogenic G protein-coupled receptor
I E Zohn, M Klinger, X Karp, et al.
Oncogene
|
May 22, 2001
The thrombin receptor, PAR-1, causes transformation by activation of Rho-mediated signaling pathways
C B Martin, G M Mahon, M B Klinger, et al.
The Prostate
|
January 1, 1988
Expression of ras proto-oncogenes in the Dunning R3327 rat prostatic adenocarcinoma system
D B Cooke, V E Quarmby, P Petrusz, et al.
Gastroenterology
|
August 28, 1999
Pharmacological inhibition of Ras-transformed epithelial cell growth is linked to down-regulation of epidermal growth factor-related peptides
N Sizemore, A D Cox, J A Barnard, et al.
The Journal of Biological Chemistry
|
April 1, 2000
Vav2 is an activator of Cdc42, Rac1, and RhoA
K Abe, K L Rossman, B Liu, et al.
Page
of 18