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International Journal of Obesity (2005)|April 19, 2006
Oxyntomodulin increases energy expenditure in addition to decreasing energy intake in overweight and obese humans: a randomised controlled trialK Wynne, A J Park, C J Small, et al.Regulatory Peptides|February 7, 2002
Chronic intraparaventricular nuclear administration of orexin A in male rats does not alter thyroid axis or uncoupling protein-1 in brown adipose tissueS H Russell, C J Small, D Sunter, et al.FEBS Letters|May 20, 2003
gamma-MSH increases intracellular cAMP accumulation and GnRH release in vitro and LH release in vivoS A Stanley, S Davies, C J Small, et al.Endocrinology|May 10, 2000
Prolactin releasing peptide (PrRP) stimulates luteinizing hormone (LH) and follicle stimulating hormone (FSH) via a hypothalamic mechanism in male ratsL J Seal, C J Small, M S Kim, et al.International Journal of Obesity and Related Metabolic Disorders : Journal of the International Association for the Study of Obesity|March 29, 2003
Chronic CNS administration of Agouti-related protein (Agrp) reduces energy expenditureC J Small, Y L Liu, S A Stanley, et al.Endocrinology|October 1, 1996
Glucagon-like peptide-1 (GLP-1) releases thyrotropin (TSH): characterization of binding sites for GLP-1 on alpha-TSH cellsS A Beak, C J Small, I Ilovaiskaia, et al.Journal of Neuroendocrinology|June 20, 2001
The central effects of orexin-A in the hypothalamic-pituitary-adrenal axis in vivo and in vitro in male ratsS H Russell, C J Small, C L Dakin, et al.Endocrinology|June 21, 2001
Melanin-concentrating hormone (MCH) suppresses thyroid stimulating hormone (TSH) release, in vivo and in vitro, via the hypothalamus and the pituitaryA R Kennedy, J F Todd, S A Stanley, et al.International Journal of Obesity (2005)|October 26, 2005
Subcutaneous administration of ghrelin stimulates energy intake in healthy lean human volunteersM R Druce, N M Neary, C J Small, et al.Endocrinology|September 21, 2001
PRL-releasing peptide inhibits food intake in male rats via the dorsomedial hypothalamic nucleus and not the paraventricular hypothalamic nucleusL J Seal, C J Small, W S Dhillo, et al.Pageof 119