C L Fasching - JoVE Visualize

Showing results (1-10 of 10) with videos related to

Pageof 1

Sort By:

Genes, Chromosomes & Cancer|April 1, 1994

Evidence that wild-type TP53, and not genes on either chromosome 1 or 11, controls the tumorigenic phenotype of the human fibrosarcoma HT1080M J Anderson, G Casey, C L Fasching, et al.

Nature Genetics|December 2, 2000

Telomere maintenance by recombination in human cellsM A Dunham, A A Neumann, C L Fasching, et al.

Genes, Chromosomes & Cancer|November 1, 1990

Irradiation microcell-mediated chromosome transfer (XMMCT): the generation of specific chromosomal arm deletionsS F Dowdy, D J Scanlon, C L Fasching, et al.

Radiation Research|July 1, 1995

Loss of a putative tumor suppressor locus after gamma-ray-induced neoplastic transformation of HeLa x skin fibroblast human cell hybridsM S Mendonca, C L Fasching, E S Srivatsan, et al.

Genes, Chromosomes & Cancer|April 1, 1994

Chromosome 13 transfer provides evidence for regulation of RB1 protein expressionM J Anderson, C L Fasching, H J Xu, et al.

Cancer Research|January 15, 1994

Analysis of the tumor suppressor activity of the K-rev-1 gene in human tumor cell linesK Y Sato, P G Polakis, H Haubruck, et al.

Proceedings of the National Academy of Sciences of the United States of America|June 25, 1996

Loss of oncogenic ras expression does not correlate with loss of tumorigenicity in human cellsR Plattner, M J Anderson, K Y Sato, et al.

Science (New York, N.Y.)|October 11, 1991

Suppression of tumorigenicity in Wilms tumor by the p15.5-p14 region of chromosome 11S F Dowdy, C L Fasching, D Araujo, et al.

Radiation Research|March 13, 1998

Loss of suppressor loci on chromosomes 11 and 14 may be required for radiation-induced neoplastic transformation of HeLa x skin fibroblast human cell hybridsM S Mendonca, K Howard, C L Fasching, et al.

Molecular and Cellular Biology|March 1, 1992

Progression of colorectal cancer is associated with multiple tumor suppressor gene defects but inhibition of tumorigenicity is accomplished by correction of any single defect via chromosome transferM C Goyette, K Cho, C L Fasching, et al.

Pageof 1

Showing results (1-10 of 10) with videos related to

Sort By:

Pageof 1

Genes, Chromosomes & Cancer|April 1, 1994

Evidence that wild-type TP53, and not genes on either chromosome 1 or 11, controls the tumorigenic phenotype of the human fibrosarcoma HT1080M J Anderson, G Casey, C L Fasching, et al.

Nature Genetics|December 2, 2000

Telomere maintenance by recombination in human cellsM A Dunham, A A Neumann, C L Fasching, et al.

Genes, Chromosomes & Cancer|November 1, 1990

Irradiation microcell-mediated chromosome transfer (XMMCT): the generation of specific chromosomal arm deletionsS F Dowdy, D J Scanlon, C L Fasching, et al.

Radiation Research|July 1, 1995

Loss of a putative tumor suppressor locus after gamma-ray-induced neoplastic transformation of HeLa x skin fibroblast human cell hybridsM S Mendonca, C L Fasching, E S Srivatsan, et al.

Genes, Chromosomes & Cancer|April 1, 1994

Chromosome 13 transfer provides evidence for regulation of RB1 protein expressionM J Anderson, C L Fasching, H J Xu, et al.

Cancer Research|January 15, 1994

Analysis of the tumor suppressor activity of the K-rev-1 gene in human tumor cell linesK Y Sato, P G Polakis, H Haubruck, et al.

Proceedings of the National Academy of Sciences of the United States of America|June 25, 1996

Loss of oncogenic ras expression does not correlate with loss of tumorigenicity in human cellsR Plattner, M J Anderson, K Y Sato, et al.

Science (New York, N.Y.)|October 11, 1991

Suppression of tumorigenicity in Wilms tumor by the p15.5-p14 region of chromosome 11S F Dowdy, C L Fasching, D Araujo, et al.

Radiation Research|March 13, 1998

Loss of suppressor loci on chromosomes 11 and 14 may be required for radiation-induced neoplastic transformation of HeLa x skin fibroblast human cell hybridsM S Mendonca, K Howard, C L Fasching, et al.

Molecular and Cellular Biology|March 1, 1992

Progression of colorectal cancer is associated with multiple tumor suppressor gene defects but inhibition of tumorigenicity is accomplished by correction of any single defect via chromosome transferM C Goyette, K Cho, C L Fasching, et al.

Pageof 1