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Health Care Management Review|January 1, 1996
Application of group process model to performance appraisal development in a CQI environmentC LaPenta, G M JacobsStem Cells (Dayton, Ohio)|May 14, 2003
A new type I IFN-mediated pathway for the rapid differentiation of monocytes into highly active dendritic cellsS M Santini, T Di Pucchio, C Lapenta, et al.The Journal of Experimental Medicine|May 17, 2000
Type I interferon as a powerful adjuvant for monocyte-derived dendritic cell development and activity in vitro and in Hu-PBL-SCID miceS M Santini, C Lapenta, M Logozzi, et al.Le Infezioni in Medicina|January 1, 1997
[Serum anti-HIV IgA in seropositive patients and in subjects at risk of HIV infection]P Filippini, M Guarino, C Lapenta, et al.Le Infezioni in Medicina|January 1, 1997
[HIV Antigens as complement fixing circulating immune complexes]P Filippini, M Guarino, C Lapenta, et al.Transplantation|March 4, 1998
Treatment of severe combined immunodeficiency mice with anti-murine granulocyte monoclonal antibody improves human leukocyte xenotransplantationS M Santini, M Spada, S Parlato, et al.Journal of Virology|July 10, 1999
Human immunodeficiency virus type 1 strains R5 and X4 induce different pathogenic effects in hu-PBL-SCID mice, depending on the state of activation/differentiation of human target cells at the time of primary infectionS Fais, C Lapenta, S M Santini, et al.European Journal of Immunology|May 6, 1999
Human intestinal lamina propria lymphocytes are naturally permissive to HIV-1 infectionC Lapenta, M Boirivant, M Marini, et al.Journal of Virology|November 13, 1998
Human lymphoblastoid CD4(+) T cells become permissive to macrophage-tropic strains of human immunodeficiency virus type 1 after passage into severe combined immunodeficient mice through in vivo upregulation of CCR5: in vivo dynamics of CD4(+) T-cell differentiation in pathogenesis of AIDSC Lapenta, S Parlato, M Spada, et al.Cell Death and Differentiation|March 14, 2000
Primary HIV-1 infection of human CD4+ T cells passaged into SCID mice leads to selection of chronically infected cells through a massive fas-mediated autocrine suicide of uninfected cellsS Parlato, S M Santini, C Lapenta, et al.Pageof 3