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Cancer Research|February 24, 2001
Bax accelerates tumorigenesis in p53-deficient miceC M Knudson, G M Johnson, Y Lin, et al.Nature Genetics|August 1, 1997
Bcl-2 and Bax function independently to regulate cell deathC M Knudson, S J KorsmeyerNature|February 13, 1997
Bax suppresses tumorigenesis and stimulates apoptosis in vivoC Yin, C M Knudson, S J Korsmeyer, et al.Investigative Ophthalmology & Visual Science|August 12, 1998
Suppression of developmental retinal cell death but not of photoreceptor degeneration in Bax-deficient miceJ Mosinger Ogilvie, T L Deckwerth, C M Knudson, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 27, 1999
Neurotrophins support the development of diverse sensory axon morphologiesS I Lentz, C M Knudson, S J Korsmeyer, et al.Nature Medicine|November 14, 1997
Apoptosis-associated signaling pathways are required for chemotherapy-mediated female germ cell destructionG I Perez, C M Knudson, L Leykin, et al.Science (New York, N.Y.)|October 6, 1995
Bax-deficient mice with lymphoid hyperplasia and male germ cell deathC M Knudson, K S Tung, W G Tourtellotte, et al.Experimental Neurology|July 31, 1998
Placement of the BCL2 family member BAX in the death pathway of sympathetic neurons activated by trophic factor deprivationT L Deckwerth, R M Easton, C M Knudson, et al.Proceedings of the National Academy of Sciences of the United States of America|March 18, 1997
bax-deficiency promotes drug resistance and oncogenic transformation by attenuating p53-dependent apoptosisM E McCurrach, T M Connor, C M Knudson, et al.Nature Medicine|December 10, 1998
Hyperglycemia induces apoptosis in pre-implantation embryos through cell death effector pathwaysK H Moley, M M Chi, C M Knudson, et al.Pageof 726