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Antioxidants & Redox Signaling|March 10, 2001
Reactive nitrogen species in colon carcinogenesisC M Payne, C Bernstein, H Bernstein, et al.Ultrastructural Pathology|July 1, 1995
Role of apoptosis in biology and pathology: resistance to apoptosis in colon carcinogenesisC M Payne, H Bernstein, C Bernstein, et al.Pharmacology|March 20, 2002
Activation of the metallothionein IIA promoter and other key stress response elements by ursodeoxycholate in HepG2 cells: relevance to the cytoprotective function of ursodeoxycholateC Bernstein, C M Payne, H Bernstein, et al.Mutation Research|January 18, 2005
Bile acids as carcinogens in human gastrointestinal cancersH Bernstein, C Bernstein, C M Payne, et al.Antioxidants & Redox Signaling|January 5, 2002
The specific NOS2 inhibitor, 1400W, sensitizes HepG2 cells to genotoxic, oxidative, xenobiotic, and endoplasmic reticulum stressesC M Payne, H Bernstein, C Bernstein, et al.Cell Death and Differentiation|October 1, 1996
Bile acid activation of the gadd153 promoter and of p53-independent apoptosis: relevance to colon cancerZ Y Zheng, H Bernstein, C Bernstein, et al.Toxicology Letters|September 21, 2000
Increased susceptibility of cells to inducible apoptosis during growth from early to late log phase: an important caveat for in vitro apoptosis researchD Washo-Stultz, C Crowley, C M Payne, et al.Leukemia & Lymphoma|September 1, 1995
Apoptosis overview emphasizing the role of oxidative stress, DNA damage and signal-transduction pathwaysC M Payne, C Bernstein, H BernsteinDiseases of the Esophagus : Official Journal of the International Society for Diseases of the Esophagus|September 21, 2006
Esophageal acid exposure at pH < or = 2 is more common in Barrett's esophagus patients and is associated with oxidative stressK Dvorak, R Fass, R Dekel, et al.Cell Biology and Toxicology|December 3, 2005
Mitochondrial perturbation attenuates bile acid-induced cytotoxicityC M Payne, C L Crowley-Weber, K Dvorak, et al.Pageof 93