Showing results (41-50 of 112) with videos related to
Sort By:
Pageof 12
Journal of Virology|October 24, 2014
Identification of residues in the human respiratory syncytial virus fusion protein that modulate fusion activity and pathogenesisAnne L Hotard, Sujin Lee, Michael G Currier, et al.The Journal of Experimental Medicine|December 10, 2017
IL-33 promotes the egress of group 2 innate lymphoid cells from the bone marrowMatthew T Stier, Jian Zhang, Kasia Goleniewska, et al.Infection and Immunity|March 9, 2016
STAT6 Signaling Attenuates Interleukin-17-Producing γδ T Cells during Acute Klebsiella pneumoniae InfectionMelissa H Bloodworth, Dawn C Newcomb, Daniel E Dulek, et al.Allergy|November 19, 2024
Endogenous Glucagon-Like Peptide-1 Receptor and Glucose-Dependent Insulinotropic Polypeptide Receptor Signaling Inhibits Aeroallergen-Induced Innate Airway InflammationShinji Toki, Masako Abney, Jian Zhang, et al.Biomolecules|August 28, 2025
Association of 17q12-q21 Asthma Risk Locus with Clinical Severity of Infant Respiratory Syncytial Virus InfectionKedir N Turi, Christopher McKennan, Christian Rosas-Salazar, et al.The Journal of Allergy and Clinical Immunology|January 18, 2011
Human TH17 cells express a functional IL-13 receptor and IL-13 attenuates IL-17A productionDawn C Newcomb, Madison G Boswell, Weisong Zhou, et al.Biorxiv : the Preprint Server for Biology|January 23, 2026
Ovarian Hormones and Obesity Drive Th17-mediated Airway Inflammation through Estrogen Receptor SignalingEmely Henriquez-Pilier, Jacqueline-Yvonne Cephus, Shelby Kuehnle, et al.American Journal of Respiratory and Critical Care Medicine|December 25, 2003
Human bronchial smooth muscle cell lines show a hypertrophic phenotype typical of severe asthmaLimei Zhou, Jing Li, Adam M Goldsmith, et al.Experimental Lung Research|March 8, 2012
Intratracheal bleomycin causes airway remodeling and airflow obstruction in miceVasiliy V Polosukhin, Amber L Degryse, Dawn C Newcomb, et al.Thorax|April 14, 2016
The histone deacetylase inhibitor trichostatin A suppresses murine innate allergic inflammation by blocking group 2 innate lymphoid cell (ILC2) activationShinji Toki, Kasia Goleniewska, Sara Reiss, et al.Pageof 12