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The Biochemical Journal|February 1, 1988
Platelet-activating factor-induced hydrolysis of phosphatidylinositol 4,5-bisphosphate stimulates the production of reactive oxygen intermediates in macrophagesS J Huang, P N Monk, C P Downes, et al.
The Journal of Biological Chemistry|June 17, 1994
A G-protein beta gamma-subunit-responsive phosphoinositide 3-kinase activity in human platelet cytosolP A Thomason, S R James, P J Casey, et al.
The EMBO Journal|December 1, 1986
The haemopoietic growth factors interleukin 3 and colony stimulating factor-1 stimulate proliferation but do not induce inositol lipid breakdown in murine bone-marrow-derived macrophagesA D Whetton, P N Monk, S D Consalvey, et al.
The Biochemical Journal|April 1, 1987
Inositol 1,2-cyclic 4,5-trisphosphate is not a product of muscarinic receptor-stimulated phosphatidylinositol 4,5-bisphosphate hydrolysis in rat parotid glandsP T Hawkins, C P Berrie, A J Morris, et al.
Growth Factors (Chur, Switzerland)|January 1, 1991
Chemotactic peptides but not macrophage colony stimulating factor effect the synthesis of inositol lipids in macrophagesS J Vallance, C H MacPhee, C P Downes, et al.
The EMBO Journal|January 1, 1987
Receptor coupled events in bradykinin action: rapid production of inositol phosphates and regulation of cytosolic free Ca2+ in a neural cell lineT R Jackson, T J Hallam, C P Downes, et al.
The Biochemical Journal|August 1, 1988
Synthesis of myo-inositol 1,3,4,5,6-pentakisphosphate from inositol phosphates generated by receptor activationL R Stephens, P T Hawkins, C J Barker, et al.
The Biochemical Journal|July 15, 1994
Phosphatidylinositol 3,4,5-trisphosphate is formed from phosphatidylinositol 4,5-bisphosphate in thrombin-stimulated plateletsA N Carter, R Huang, A Sorisky, et al.
British Journal of Pharmacology|October 29, 2000
Activation of phospholipase D by metabotropic glutamate receptor agonists in rat cerebrocortical synaptosomesT Shinomura, E del Río, K C Breen, et al.
The Biochemical Journal|March 4, 2000
Analysis of the cellular functions of PTEN using catalytic domain and C-terminal mutations: differential effects of C-terminal deletion on signalling pathways downstream of phosphoinositide 3-kinaseN R Leslie, A Gray, I Pass, et al.
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