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C Pronk

Showing results (81-90 of 131) with videos related to

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Bone Marrow Transplantation|January 20, 2021
Hematopoietic cell transplant in pediatric acute myeloid leukemia after similar upfront therapy; a comparison of conditioning regimensA B Versluys, J J Boelens, C Pronk, et al.
British Journal of Cancer|February 12, 1998
Phase I study on docetaxel and ifosfamide in patients with advanced solid tumoursL C Pronk, D Schrijvers, J H Schellens, et al.
Neurology|May 12, 2004
Arg113His mutation in eIF2Bepsilon as cause of leukoencephalopathy in adultsM S van der Knaap, P A J Leegwater, C G M van Berkel, et al.
Cancer|March 1, 1987
Clinical significance of pepsinogen A isozymogens, serum pepsinogen A and C levels, and serum gastrin levelsB D Westerveld, G Pals, C B Lamers, et al.
Transplant International : Official Journal of the European Society for Organ Transplantation|November 14, 2017
"What if this is my chance to save my life?" A semistructured interview study on the motives and experiences of end-stage renal disease patients who engaged in public solicitation of a living kidney donorMathilde C Pronk, Dorthe Slaats, Willij C Zuidema, et al.
Neurology|January 28, 2004
Multiparametric MRI in a patient with adult-onset leukoencephalopathy with vanishing white matterA Gallo, M A Rocca, A Falini, et al.
American Journal of Human Genetics|August 12, 1999
The gene for leukoencephalopathy with vanishing white matter is located on chromosome 3q27P A Leegwater, A A Könst, B Kuyt, et al.
Human Genetics|January 1, 1984
Familial benign hypercalcaemia (FBH; McK. No. 14598, 1983): linkage studies in a large Dutch familyF H Menko, O L Bijvoet, P Meera Khan, et al.
Annals of Neurology|February 9, 2002
Mutations in each of the five subunits of translation initiation factor eIF2B can cause leukoencephalopathy with vanishing white matterMarjo S van der Knaap, Peter A J Leegwater, Andrea A M Könst, et al.
Human Genetics|May 1, 1989
Family and population studies on the human pepsinogen A multigene familyJ P Bebelman, M P Evers, B Zelle, et al.
Pageof 14

Showing results (81-90 of 131) with videos related to

Sort By:
Pageof 14
Bone Marrow Transplantation|January 20, 2021
Hematopoietic cell transplant in pediatric acute myeloid leukemia after similar upfront therapy; a comparison of conditioning regimensA B Versluys, J J Boelens, C Pronk, et al.
British Journal of Cancer|February 12, 1998
Phase I study on docetaxel and ifosfamide in patients with advanced solid tumoursL C Pronk, D Schrijvers, J H Schellens, et al.
Neurology|May 12, 2004
Arg113His mutation in eIF2Bepsilon as cause of leukoencephalopathy in adultsM S van der Knaap, P A J Leegwater, C G M van Berkel, et al.
Cancer|March 1, 1987
Clinical significance of pepsinogen A isozymogens, serum pepsinogen A and C levels, and serum gastrin levelsB D Westerveld, G Pals, C B Lamers, et al.
Transplant International : Official Journal of the European Society for Organ Transplantation|November 14, 2017
"What if this is my chance to save my life?" A semistructured interview study on the motives and experiences of end-stage renal disease patients who engaged in public solicitation of a living kidney donorMathilde C Pronk, Dorthe Slaats, Willij C Zuidema, et al.
Neurology|January 28, 2004
Multiparametric MRI in a patient with adult-onset leukoencephalopathy with vanishing white matterA Gallo, M A Rocca, A Falini, et al.
American Journal of Human Genetics|August 12, 1999
The gene for leukoencephalopathy with vanishing white matter is located on chromosome 3q27P A Leegwater, A A Könst, B Kuyt, et al.
Human Genetics|January 1, 1984
Familial benign hypercalcaemia (FBH; McK. No. 14598, 1983): linkage studies in a large Dutch familyF H Menko, O L Bijvoet, P Meera Khan, et al.
Annals of Neurology|February 9, 2002
Mutations in each of the five subunits of translation initiation factor eIF2B can cause leukoencephalopathy with vanishing white matterMarjo S van der Knaap, Peter A J Leegwater, Andrea A M Könst, et al.
Human Genetics|May 1, 1989
Family and population studies on the human pepsinogen A multigene familyJ P Bebelman, M P Evers, B Zelle, et al.
Pageof 14