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The Quarterly Journal of Medicine
|
May 1, 1985
The pathogenesis of renal osteodystrophy: role of vitamin D, aluminium, parathyroid hormone, calcium and phosphorus
C R Dunstan, E Hills, A W Norman, et al.
Mineral and Electrolyte Metabolism
|
January 1, 1985
Treatment of hemodialysis bone disease with 24,25-(OH)2D3 and 1,25-(OH)2D3 alone or in combination
C R Dunstan, E Hills, A W Norman, et al.
The Journal of Cell Biology
|
May 4, 1999
The ligand for osteoprotegerin (OPGL) directly activates mature osteoclasts
T L Burgess, Y Qian, S Kaufman, et al.
The Journal of Experimental Medicine
|
August 22, 2000
Osteoprotegerin reverses osteoporosis by inhibiting endosteal osteoclasts and prevents vascular calcification by blocking a process resembling osteoclastogenesis
H Min, S Morony, I Sarosi, et al.
Genes & Development
|
June 6, 1998
osteoprotegerin-deficient mice develop early onset osteoporosis and arterial calcification
N Bucay, I Sarosi, C R Dunstan, et al.
Arthritis and Rheumatism
|
June 1, 2006
The ratio of circulating osteoprotegerin to RANKL in early rheumatoid arthritis predicts later joint destruction
P P Geusens, R B M Landewé, P Garnero, et al.
Blood
|
December 12, 2001
Osteoprotegerin inhibits the development of osteolytic bone disease in multiple myeloma
P I Croucher, C M Shipman, J Lippitt, et al.
Nature
|
February 9, 1999
OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesis
Y Y Kong, H Yoshida, I Sarosi, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
March 31, 1999
Tumor necrosis factor receptor family member RANK mediates osteoclast differentiation and activation induced by osteoprotegerin ligand
H Hsu, D L Lacey, C R Dunstan, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
March 4, 2000
RANK is the intrinsic hematopoietic cell surface receptor that controls osteoclastogenesis and regulation of bone mass and calcium metabolism
J Li, I Sarosi, X Q Yan, et al.
Page
of 9
Search research articles
Search
Showing results (71-80 of 83) with videos related to
Sort By:
Page
of 9
The Quarterly Journal of Medicine
|
May 1, 1985
The pathogenesis of renal osteodystrophy: role of vitamin D, aluminium, parathyroid hormone, calcium and phosphorus
C R Dunstan, E Hills, A W Norman, et al.
Mineral and Electrolyte Metabolism
|
January 1, 1985
Treatment of hemodialysis bone disease with 24,25-(OH)2D3 and 1,25-(OH)2D3 alone or in combination
C R Dunstan, E Hills, A W Norman, et al.
The Journal of Cell Biology
|
May 4, 1999
The ligand for osteoprotegerin (OPGL) directly activates mature osteoclasts
T L Burgess, Y Qian, S Kaufman, et al.
The Journal of Experimental Medicine
|
August 22, 2000
Osteoprotegerin reverses osteoporosis by inhibiting endosteal osteoclasts and prevents vascular calcification by blocking a process resembling osteoclastogenesis
H Min, S Morony, I Sarosi, et al.
Genes & Development
|
June 6, 1998
osteoprotegerin-deficient mice develop early onset osteoporosis and arterial calcification
N Bucay, I Sarosi, C R Dunstan, et al.
Arthritis and Rheumatism
|
June 1, 2006
The ratio of circulating osteoprotegerin to RANKL in early rheumatoid arthritis predicts later joint destruction
P P Geusens, R B M Landewé, P Garnero, et al.
Blood
|
December 12, 2001
Osteoprotegerin inhibits the development of osteolytic bone disease in multiple myeloma
P I Croucher, C M Shipman, J Lippitt, et al.
Nature
|
February 9, 1999
OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesis
Y Y Kong, H Yoshida, I Sarosi, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
March 31, 1999
Tumor necrosis factor receptor family member RANK mediates osteoclast differentiation and activation induced by osteoprotegerin ligand
H Hsu, D L Lacey, C R Dunstan, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
March 4, 2000
RANK is the intrinsic hematopoietic cell surface receptor that controls osteoclastogenesis and regulation of bone mass and calcium metabolism
J Li, I Sarosi, X Q Yan, et al.
Page
of 9