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C S Apstein

Showing results (91-100 of 123) with videos related to

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The American Journal of Physiology|December 9, 1998
Increased calcium loading and inotropy without greater cell death in hypoxic rat cardiomyocytesR P Kondo, C S Apstein, F R Eberli, et al.
Cardiovascular Drugs and Therapy|May 1, 1996
Beneficial effects of felodipine on myocardial and coronary function during low-flow ischemia and reperfusionE A Bernstein, F R Eberli, A M Silverman, et al.
Hypertension (Dallas, Tex. : 1979)|May 20, 2000
Comparison of hearts with 2 types of pressure-overload left ventricular hypertrophyK W Saupe, C C Lim, J S Ingwall, et al.
Circulation|June 11, 1976
Experimental myocardial infarction. XIII. Sequential changes in left ventricular pressure-length relationships in the acute phaseF A Pirzada, E A Ekong, P S Vokonas, et al.
The Journal of Clinical Investigation|December 1, 1991
A single myocardial stretch or decreased systolic fiber shortening stimulates the expression of heat shock protein 70 in the isolated, erythrocyte-perfused rabbit heartA A Knowlton, F R Eberli, P Brecher, et al.
The American Journal of Physiology|April 1, 1985
Effect of erythrocyte storage and oxyhemoglobin affinity changes on cardiac functionC S Apstein, R C Dennis, L Briggs, et al.
The Journal of Clinical Investigation|April 1, 1992
Rapid expression of fibronectin in the rabbit heart after myocardial infarction with and without reperfusionA A Knowlton, C M Connelly, G M Romo, et al.
Cardiovascular Drugs and Therapy|April 1, 1992
Regression of left ventricular hypertrophy in "previously untreated" hypertensive blacks after 6 months of blood pressure reduction with alpha- and beta-adrenergic blockade and thiazide therapyE Foster, J F Plehn, S A Bernard, et al.
Cardiovascular Research|June 6, 1998
Early changes in excitation-contraction coupling: transition from compensated hypertrophy to failure in Dahl salt-sensitive rat myocytesK Nagata, R Liao, F R Eberli, et al.
The Journal of Clinical Investigation|December 1, 1990
Increased rat cardiac angiotensin converting enzyme activity and mRNA expression in pressure overload left ventricular hypertrophy. Effects on coronary resistance, contractility, and relaxationH Schunkert, V J Dzau, S S Tang, et al.
Pageof 13

Showing results (91-100 of 123) with videos related to

Sort By:
Pageof 13
The American Journal of Physiology|December 9, 1998
Increased calcium loading and inotropy without greater cell death in hypoxic rat cardiomyocytesR P Kondo, C S Apstein, F R Eberli, et al.
Cardiovascular Drugs and Therapy|May 1, 1996
Beneficial effects of felodipine on myocardial and coronary function during low-flow ischemia and reperfusionE A Bernstein, F R Eberli, A M Silverman, et al.
Hypertension (Dallas, Tex. : 1979)|May 20, 2000
Comparison of hearts with 2 types of pressure-overload left ventricular hypertrophyK W Saupe, C C Lim, J S Ingwall, et al.
Circulation|June 11, 1976
Experimental myocardial infarction. XIII. Sequential changes in left ventricular pressure-length relationships in the acute phaseF A Pirzada, E A Ekong, P S Vokonas, et al.
The Journal of Clinical Investigation|December 1, 1991
A single myocardial stretch or decreased systolic fiber shortening stimulates the expression of heat shock protein 70 in the isolated, erythrocyte-perfused rabbit heartA A Knowlton, F R Eberli, P Brecher, et al.
The American Journal of Physiology|April 1, 1985
Effect of erythrocyte storage and oxyhemoglobin affinity changes on cardiac functionC S Apstein, R C Dennis, L Briggs, et al.
The Journal of Clinical Investigation|April 1, 1992
Rapid expression of fibronectin in the rabbit heart after myocardial infarction with and without reperfusionA A Knowlton, C M Connelly, G M Romo, et al.
Cardiovascular Drugs and Therapy|April 1, 1992
Regression of left ventricular hypertrophy in "previously untreated" hypertensive blacks after 6 months of blood pressure reduction with alpha- and beta-adrenergic blockade and thiazide therapyE Foster, J F Plehn, S A Bernard, et al.
Cardiovascular Research|June 6, 1998
Early changes in excitation-contraction coupling: transition from compensated hypertrophy to failure in Dahl salt-sensitive rat myocytesK Nagata, R Liao, F R Eberli, et al.
The Journal of Clinical Investigation|December 1, 1990
Increased rat cardiac angiotensin converting enzyme activity and mRNA expression in pressure overload left ventricular hypertrophy. Effects on coronary resistance, contractility, and relaxationH Schunkert, V J Dzau, S S Tang, et al.
Pageof 13