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C S Apstein

Showing results (41-50 of 123) with videos related to

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Recent Advances in Studies on Cardiac Structure and Metabolism|January 1, 1975
Factors influencing tolerance of cardiac muscle to hypoxiaO H Bing, C S Apstein, W W Brooks
Circulation|December 31, 1997
Ischemic preconditioning does not protect against contractile dysfunction in the presence of residual flow: studies in the isolated, blood-perfused rat heartA C Cave, A S Silverman, C S Apstein
The American Journal of Physiology|December 11, 1978
Acute cardiac ischemia and reperfusion: contractility, relaxation, and glycolysisC S Apstein, L Deckelbaum, L Hagopian, et al.
The American Journal of Physiology|November 24, 1999
Impaired lusitropy-frequency in the aging mouse: role of Ca(2+)-handling proteins and effects of isoproterenolC C Lim, R Liao, N Varma, et al.
Journal of Molecular and Cellular Cardiology|June 1, 1989
Leakage of heart fatty acid binding protein with ischemia and reperfusion in the ratA A Knowlton, C S Apstein, R Saouf, et al.
The Journal of Clinical Investigation|July 1, 1981
Comparison of acute alterations in left ventricular relaxation and diastolic chamber stiffness induced by hypoxia and ischemia. Role of myocardial oxygen supply-demand imbalanceT Serizawa, W M Vogel, C S Apstein, et al.
The Journal of Clinical Investigation|February 1, 1992
Exacerbation of ischemic dysfunction by angiotensin II in red cell-perfused rabbit hearts. Effects on coronary flow, contractility, and high-energy phosphate metabolismT Mochizuki, F R Eberli, C S Apstein, et al.
The American Journal of Physiology|November 1, 1982
Movement of necrotic wavefront after coronary artery occlusion in rabbitC Connelly, W M Vogel, Y M Hernandez, et al.
Circulation Research|May 1, 1992
Exacerbation of left ventricular ischemic diastolic dysfunction by pressure-overload hypertrophy. Modification by specific inhibition of cardiac angiotensin converting enzymeF R Eberli, C S Apstein, S Ngoy, et al.
Progress in Clinical and Biological Research|January 1, 1989
Studies of the mechanism of the vasoconstrictor activity of stroma-free hemoglobin in the isolated perfused rat kidney and rabbit heartW Lieberthal, W M Vogel, C S Apstein, et al.
Pageof 13

Showing results (41-50 of 123) with videos related to

Sort By:
Pageof 13
Recent Advances in Studies on Cardiac Structure and Metabolism|January 1, 1975
Factors influencing tolerance of cardiac muscle to hypoxiaO H Bing, C S Apstein, W W Brooks
Circulation|December 31, 1997
Ischemic preconditioning does not protect against contractile dysfunction in the presence of residual flow: studies in the isolated, blood-perfused rat heartA C Cave, A S Silverman, C S Apstein
The American Journal of Physiology|December 11, 1978
Acute cardiac ischemia and reperfusion: contractility, relaxation, and glycolysisC S Apstein, L Deckelbaum, L Hagopian, et al.
The American Journal of Physiology|November 24, 1999
Impaired lusitropy-frequency in the aging mouse: role of Ca(2+)-handling proteins and effects of isoproterenolC C Lim, R Liao, N Varma, et al.
Journal of Molecular and Cellular Cardiology|June 1, 1989
Leakage of heart fatty acid binding protein with ischemia and reperfusion in the ratA A Knowlton, C S Apstein, R Saouf, et al.
The Journal of Clinical Investigation|July 1, 1981
Comparison of acute alterations in left ventricular relaxation and diastolic chamber stiffness induced by hypoxia and ischemia. Role of myocardial oxygen supply-demand imbalanceT Serizawa, W M Vogel, C S Apstein, et al.
The Journal of Clinical Investigation|February 1, 1992
Exacerbation of ischemic dysfunction by angiotensin II in red cell-perfused rabbit hearts. Effects on coronary flow, contractility, and high-energy phosphate metabolismT Mochizuki, F R Eberli, C S Apstein, et al.
The American Journal of Physiology|November 1, 1982
Movement of necrotic wavefront after coronary artery occlusion in rabbitC Connelly, W M Vogel, Y M Hernandez, et al.
Circulation Research|May 1, 1992
Exacerbation of left ventricular ischemic diastolic dysfunction by pressure-overload hypertrophy. Modification by specific inhibition of cardiac angiotensin converting enzymeF R Eberli, C S Apstein, S Ngoy, et al.
Progress in Clinical and Biological Research|January 1, 1989
Studies of the mechanism of the vasoconstrictor activity of stroma-free hemoglobin in the isolated perfused rat kidney and rabbit heartW Lieberthal, W M Vogel, C S Apstein, et al.
Pageof 13