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Multiple Sclerosis (Houndmills, Basingstoke, England)|June 21, 2008
Fatal neurogenic pulmonary edema in a patient with progressive multiple sclerosisS Bramow, J C Faber-Rod, C Jacobsen, et al.Multiple Sclerosis (Houndmills, Basingstoke, England)|October 31, 2007
Fulminant central nervous system demyelination associated with interferon-alpha therapy and hepatitis C virus infectionR Höftberger, F Garzuly, H P Dienes, et al.Immunobiology|July 1, 1988
Expression of the VEP13 antigen (CD16) on native human alveolar macrophages and cultured blood monocytesI Baumgartner, O Scheiner, C Holzinger, et al.The Journal of Biological Chemistry|November 21, 1998
The low density lipoprotein receptor gene family. Differential expression of two alpha2-macroglobulin receptors in the brainW Stockinger, E Hengstschläger-Ottnad, S Novak, et al.Journal of Neuropathology and Experimental Neurology|January 1, 1989
An antigenic profile of Lewy bodies: immunocytochemical indication for protein phosphorylation and ubiquitinationC Bancher, H Lassmann, H Budka, et al.Multiple Sclerosis (Houndmills, Basingstoke, England)|February 15, 2014
TIRC7 and HLA-DR axis contributes to inflammation in multiple sclerosisJ M Frischer, M Reindl, B Künz, et al.Journal of Neuroimmunology|December 1, 1995
The N-terminal domain of the myelin oligodendrocyte glycoprotein (MOG) induces acute demyelinating experimental autoimmune encephalomyelitis in the Lewis ratM Adelmann, J Wood, I Benzel, et al.European Journal of Immunology|February 17, 1998
Myelin oligodendrocyte glycoprotein-induced autoimmune encephalomyelitis is chronic/relapsing in perforin knockout mice, but monophasic in Fas- and Fas ligand-deficient lpr and gld miceU Malipiero, K Frei, K S Spanaus, et al.European Journal of Immunology|June 1, 1993
T cells specific for the myelin oligodendrocyte glycoprotein mediate an unusual autoimmune inflammatory response in the central nervous systemC Linington, T Berger, L Perry, et al.Proceedings of the National Academy of Sciences of the United States of America|November 1, 2001
Enterocolitis induced by autoimmune targeting of enteric glial cells: a possible mechanism in Crohn's disease?A Cornet, T C Savidge, J Cabarrocas, et al.Pageof 29