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Oncogene|December 26, 2001
Loss of p16 pathways stabilizes EWS/FLI1 expression and complements EWS/FLI1 mediated transformationB Deneen, C T DennyProceedings of the National Academy of Sciences of the United States of America|November 1, 1990
Streamlined approach to creating yeast artificial chromosome libraries from specialized cell sourcesJ M Feingold, S D Ogden, C T DennyMolecular and Cellular Biology|April 1, 1991
Characterization of the BCR promoter in Philadelphia chromosome-positive and -negative cell linesN P Shah, O N Witte, C T DennyOncogene|October 19, 1999
Divergent Ewing's sarcoma EWS/ETS fusions confer a common tumorigenic phenotype on NIH3T3 cellsA D Thompson, M A Teitell, A Arvand, et al.Cancer Research|June 30, 2001
The COOH-terminal domain of FLI-1 is necessary for full tumorigenesis and transcriptional modulation by EWS/FLI-1A Arvand, S M Welford, M A Teitell, et al.Genetic Analysis, Techniques and Applications|June 1, 1992
Creation of a yeast artificial chromosome fragmentation vector based on lysine-2B C Lewis, N P Shah, B S Braun, et al.Oncogene|February 2, 1995
Multiple domains mediate transformation by the Ewing's sarcoma EWS/FLI-1 fusion geneS L Lessnick, B S Braun, C T Denny, et al.Molecular and Cellular Biology|November 1, 1985
Burkitt lymphoma cell line carrying a variant translocation creates new DNA at the breakpoint and violates the hierarchy of immunoglobulin gene rearrangementC T Denny, G F Hollis, I T Magrath, et al.The Journal of Biological Chemistry|September 13, 2001
DNA binding domain-independent pathways are involved in EWS/FLI1-mediated oncogenesisS M Welford, S P Hebert, B Deneen, et al.Pageof 3