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Callie S Kwartler

Showing results (1-10 of 33) with videos related to

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Arteriosclerosis, Thrombosis, and Vascular Biology|May 2, 2024
Use of iPSC-Derived Smooth Muscle Cells to Model Physiology and PathologyCallie S Kwartler, Jose Emiliano Esparza Pinelo
American Journal of Medical Genetics. Part A|October 13, 2021
Aortic root dilatation and dilated cardiomyopathy in an adult with Tatton-Brown-Rahman syndromeAlana C Cecchi, Amier Haidar, Isabella Marin, et al.
Genetics in Medicine : Official Journal of the American College of Medical Genetics|February 5, 2010
Genetic variants promoting smooth muscle cell proliferation can result in diffuse and diverse vascular diseases: evidence for a hyperplastic vasculomyopathyDianna M Milewicz, Callie S Kwartler, Christina L Papke, et al.
Development (Cambridge, England)|June 17, 2020
Stromal β-catenin activation impacts nephron progenitor differentiation in the developing kidney and may contribute to Wilms tumorKeri A Drake, Christopher P Chaney, Amrita Das, et al.
JCI Insight|November 8, 2023
Pericentrin deficiency in smooth muscle cells augments atherosclerosis through HSF1-driven cholesterol biosynthesis and PERK activationSuravi Majumder, Abhijnan Chattopadhyay, Jamie M Wright, et al.
Genetics|January 3, 2012
Notch signaling is antagonized by SAO-1, a novel GYF-domain protein that interacts with the E3 ubiquitin ligase SEL-10 in Caenorhabditis elegansValerie A Hale, Evan L Guiney, Lindsey Y Goldberg, et al.
Biology Methods & Protocols|May 7, 2025
Measurement of oxygen consumption rate in mouse aortic tissueZhen Zhou, Ripon Sarkar, Jose Emiliano Esparza Pinelo, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|January 4, 2019
Reversal of Aortic Enlargement Induced by Increased Biomechanical Forces Requires AT1R Inhibition in Conjunction With AT2R ActivationZhen Zhou, Andrew M Peters, Shanzhi Wang, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|October 8, 2020
Cholesterol-Induced Phenotypic Modulation of Smooth Muscle Cells to Macrophage/Fibroblast-like Cells Is Driven by an Unfolded Protein ResponseAbhijnan Chattopadhyay, Callie S Kwartler, Kaveeta Kaw, et al.
American Journal of Medical Genetics. Part A|October 22, 2019
Grange syndrome due to homozygous YY1AP1 missense rare variantsIsabella V Ciuffetelli Alamo, Callie S Kwartler, Ellen R Regalado, et al.
Pageof 4

Showing results (1-10 of 33) with videos related to

Sort By:
Pageof 4
Arteriosclerosis, Thrombosis, and Vascular Biology|May 2, 2024
Use of iPSC-Derived Smooth Muscle Cells to Model Physiology and PathologyCallie S Kwartler, Jose Emiliano Esparza Pinelo
American Journal of Medical Genetics. Part A|October 13, 2021
Aortic root dilatation and dilated cardiomyopathy in an adult with Tatton-Brown-Rahman syndromeAlana C Cecchi, Amier Haidar, Isabella Marin, et al.
Genetics in Medicine : Official Journal of the American College of Medical Genetics|February 5, 2010
Genetic variants promoting smooth muscle cell proliferation can result in diffuse and diverse vascular diseases: evidence for a hyperplastic vasculomyopathyDianna M Milewicz, Callie S Kwartler, Christina L Papke, et al.
Development (Cambridge, England)|June 17, 2020
Stromal β-catenin activation impacts nephron progenitor differentiation in the developing kidney and may contribute to Wilms tumorKeri A Drake, Christopher P Chaney, Amrita Das, et al.
JCI Insight|November 8, 2023
Pericentrin deficiency in smooth muscle cells augments atherosclerosis through HSF1-driven cholesterol biosynthesis and PERK activationSuravi Majumder, Abhijnan Chattopadhyay, Jamie M Wright, et al.
Genetics|January 3, 2012
Notch signaling is antagonized by SAO-1, a novel GYF-domain protein that interacts with the E3 ubiquitin ligase SEL-10 in Caenorhabditis elegansValerie A Hale, Evan L Guiney, Lindsey Y Goldberg, et al.
Biology Methods & Protocols|May 7, 2025
Measurement of oxygen consumption rate in mouse aortic tissueZhen Zhou, Ripon Sarkar, Jose Emiliano Esparza Pinelo, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|January 4, 2019
Reversal of Aortic Enlargement Induced by Increased Biomechanical Forces Requires AT1R Inhibition in Conjunction With AT2R ActivationZhen Zhou, Andrew M Peters, Shanzhi Wang, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|October 8, 2020
Cholesterol-Induced Phenotypic Modulation of Smooth Muscle Cells to Macrophage/Fibroblast-like Cells Is Driven by an Unfolded Protein ResponseAbhijnan Chattopadhyay, Callie S Kwartler, Kaveeta Kaw, et al.
American Journal of Medical Genetics. Part A|October 22, 2019
Grange syndrome due to homozygous YY1AP1 missense rare variantsIsabella V Ciuffetelli Alamo, Callie S Kwartler, Ellen R Regalado, et al.
Pageof 4