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Cell and Tissue Research|May 18, 2012
Dual role of lipoproteins in endothelial cell dysfunction in atherosclerosisCamelia S Stancu, Laura Toma, Anca V SimaCell and Tissue Research|September 18, 2008
Vascular endothelium in atherosclerosisAnca V Sima, Camelia S Stancu, Maya SimionescuBiochemical and Biophysical Research Communications|July 7, 2011
Anti-oxidant and anti-inflammatory mechanisms of amlodipine action to improve endothelial cell dysfunction induced by irreversibly glycated LDLLaura Toma, Camelia S Stancu, Gabriela M Sanda, et al.Molecular and Cellular Biochemistry|May 22, 2016
Glycated LDL increase VCAM-1 expression and secretion in endothelial cells and promote monocyte adhesion through mechanisms involving endoplasmic reticulum stressLaura Toma, Gabriela M Sanda, Mariana Deleanu, et al.International Journal of Molecular Sciences|August 12, 2023
Elevated Levels of Circulating lncRNAs LIPCAR and MALAT1 Predict an Unfavorable Outcome in Acute Coronary Syndrome PatientsTeodora Barbalata, Loredan S Niculescu, Camelia S Stancu, et al.Biochemical and Biophysical Research Communications|October 24, 2009
Irreversibly glycated LDL induce oxidative and inflammatory state in human endothelial cells; added effect of high glucoseLaura Toma, Camelia S Stancu, Gabriela M Botez, et al.Molecular Biology Reports|August 19, 2021
MiR-223-3p levels in the plasma and atherosclerotic plaques are increased in aged patients with carotid artery stenosis; association with HDL-related proteinsTeodora Barbalata, Oriana E Moraru, Camelia S Stancu, et al.Journal of Cellular Biochemistry|June 25, 2016
Oxidized LDL-Exposed Human Macrophages Display Increased MMP-9 Expression and Secretion Mediated by Endoplasmic Reticulum StressGabriela M Sanda, Mariana Deleanu, Laura Toma, et al.Journal of Cellular and Molecular Medicine|October 13, 2009
Effect of irreversibly glycated LDL in human vascular smooth muscle cells: lipid loading, oxidative and inflammatory stressAnca V Sima, Gabriela M Botez, Camelia S Stancu, et al.Biofactors (Oxford, England)|July 29, 2017
Caffeic acid attenuates the inflammatory stress induced by glycated LDL in human endothelial cells by mechanisms involving inhibition of AGE-receptor, oxidative, and endoplasmic reticulum stressLaura Toma, Gabriela M Sanda, Loredan S Niculescu, et al.Pageof 3