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Cancer Drug Resistance (Alhambra, Calif.)|September 30, 2022
Base excision repair accessory factors in senescence avoidance and resistance to treatmentsElise Vickridge, Camila C F Faraco, Alain NepveuCancers|July 2, 2021
CUT Domain Proteins in DNA Repair and CancerZubaidah M Ramdzan, Elise Vickridge, Camila C F Faraco, et al.Journal of Molecular Biology|January 15, 2021
CUT Domains Stimulate Pol β Enzymatic Activities to Accelerate Completion of Base Excision RepairZubaidah M Ramdzan, Elise Vickridge, Li Li, et al.NAR Cancer|October 3, 2022
The DNA repair function of BCL11A suppresses senescence and promotes continued proliferation of triple-negative breast cancer cellsElise Vickridge, Camila C F Faraco, Payman S Tehrani, et al.Nucleic Acids Research|November 13, 2023
The function of BCL11B in base excision repair contributes to its dual role as an oncogene and a haplo-insufficient tumor suppressor geneElise Vickridge, Camila C F Faraco, Fanny Lo, et al.Nucleic Acids Research|July 30, 2025
The function of MYC in base excision repair protects against RAS-induced senescenceCamila C F Faraco, Wanting Zhu, Anne-Marie Fortier, et al.Pageof 1