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Mbio|July 7, 2016
Identification and Characterization of a Candidate Wolbachia pipientis Type IV Effector That Interacts with the Actin CytoskeletonKathy B Sheehan, MaryAnn Martin, Cammie F Lesser, et al.
Nature Methods|June 2, 2009
Protein interaction platforms: visualization of interacting proteins in yeastAlexa M Schmitz, Monica F Morrison, Akochi O Agunwamba, et al.
Science (New York, N.Y.)|March 16, 2019
Functional degradation: A mechanism of NLRP1 inflammasome activation by diverse pathogen enzymesAndrew Sandstrom, Patrick S Mitchell, Lisa Goers, et al.
Biochemistry|April 22, 2026
Profiling Selectivity for the Shigella Virulence Factor OspFNicholas P McCurtin, Ariana Gazaferi, Noah D Novick, et al.
Plos Pathogens|December 2, 2008
GEF-H1 mediated control of NOD1 dependent NF-kappaB activation by Shigella effectorsAtsuko Fukazawa, Carmen Alonso, Kiyotaka Kurachi, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 15, 2016
The type III secretion system apparatus determines the intracellular niche of bacterial pathogensJuan Du, Analise Z Reeves, Jessica A Klein, et al.
ACS Synthetic Biology|April 9, 2015
Engineering Escherichia coli into a protein delivery system for mammalian cellsAnalise Z Reeves, William E Spears, Juan Du, et al.
Biorxiv : the Preprint Server for Biology|February 20, 2025
Shigella OspF blocks rapid p38-dependent priming of the NAIP-NLRC4 inflammasomeElizabeth A Turcotte, Kyungsub Kim, Kevin D Eislmayr, et al.
Ebiomedicine|August 31, 2024
Gut bacterial type III secretion systems aggravate colitis in mice and serve as biomarkers of Crohn's diseaseJun Xu, Peijie Li, Zhenye Li, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 14, 2026
Shigella OspF blocks rapid p38-dependent priming of the NAIP-NLRC4 inflammasomeElizabeth A Turcotte, Kyungsub Kim, Kevin D Eislmayr, et al.
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