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Candice M Thomas

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Clinical Science (London, England : 1979)|November 3, 2012
Direct renin inhibition prevents cardiac dysfunction in a diabetic mouse model: comparison with an angiotensin receptor antagonist and angiotensin-converting enzyme inhibitorCandice M Thomas, Qian Chen Yong, Rachid Seqqat, et al.
Hypertension (Dallas, Tex. : 1979)|October 10, 2012
An elevation in physical coupling of type 1 inositol 1,4,5-trisphosphate (IP3) receptors to transient receptor potential 3 (TRPC3) channels constricts mesenteric arteries in genetic hypertensionAdebowale Adebiyi, Candice M Thomas-Gatewood, M Dennis Leo, et al.
Circulation Research|October 3, 2009
Smooth muscle cell alpha2delta-1 subunits are essential for vasoregulation by CaV1.2 channelsJohn P Bannister, Adebowale Adebiyi, Guiling Zhao, et al.
Circulation Research|August 9, 2012
TMEM16A/ANO1 channels contribute to the myogenic response in cerebral arteriesSimon Bulley, Zachary P Neeb, Sarah K Burris, et al.
American Journal of Physiology. Heart and Circulatory Physiology|January 17, 2012
Cardiac-specific genetic inhibition of nuclear factor-κB prevents right ventricular hypertrophy induced by monocrotalineSandeep Kumar, Chuanyu Wei, Candice M Thomas, et al.
Journal of Molecular and Cellular Cardiology|August 20, 2016
Loss of myocardial retinoic acid receptor α induces diastolic dysfunction by promoting intracellular oxidative stress and calcium mishandling in adult miceSen Zhu, Rakeshwar S Guleria, Candice M Thomas, et al.
American Journal of Physiology. Heart and Circulatory Physiology|August 3, 2014
Cardiac-specific suppression of NF-κB signaling prevents diabetic cardiomyopathy via inhibition of the renin-angiotensin systemCandice M Thomas, Qian Chen Yong, Rodolfo M Rosa, et al.
Circulation. Heart Failure|March 6, 2015
Phosphorylation of cardiac Myosin-binding protein-C is a critical mediator of diastolic functionPaola C Rosas, Yang Liu, Mohamed I Abdalla, et al.
Pageof 2

Showing results (11-20 of 18) with videos related to

Sort By:
Pageof 2
You have reached the last page of results.This site can display upto 18 results.
Clinical Science (London, England : 1979)|November 3, 2012
Direct renin inhibition prevents cardiac dysfunction in a diabetic mouse model: comparison with an angiotensin receptor antagonist and angiotensin-converting enzyme inhibitorCandice M Thomas, Qian Chen Yong, Rachid Seqqat, et al.
Hypertension (Dallas, Tex. : 1979)|October 10, 2012
An elevation in physical coupling of type 1 inositol 1,4,5-trisphosphate (IP3) receptors to transient receptor potential 3 (TRPC3) channels constricts mesenteric arteries in genetic hypertensionAdebowale Adebiyi, Candice M Thomas-Gatewood, M Dennis Leo, et al.
Circulation Research|October 3, 2009
Smooth muscle cell alpha2delta-1 subunits are essential for vasoregulation by CaV1.2 channelsJohn P Bannister, Adebowale Adebiyi, Guiling Zhao, et al.
Circulation Research|August 9, 2012
TMEM16A/ANO1 channels contribute to the myogenic response in cerebral arteriesSimon Bulley, Zachary P Neeb, Sarah K Burris, et al.
American Journal of Physiology. Heart and Circulatory Physiology|January 17, 2012
Cardiac-specific genetic inhibition of nuclear factor-κB prevents right ventricular hypertrophy induced by monocrotalineSandeep Kumar, Chuanyu Wei, Candice M Thomas, et al.
Journal of Molecular and Cellular Cardiology|August 20, 2016
Loss of myocardial retinoic acid receptor α induces diastolic dysfunction by promoting intracellular oxidative stress and calcium mishandling in adult miceSen Zhu, Rakeshwar S Guleria, Candice M Thomas, et al.
American Journal of Physiology. Heart and Circulatory Physiology|August 3, 2014
Cardiac-specific suppression of NF-κB signaling prevents diabetic cardiomyopathy via inhibition of the renin-angiotensin systemCandice M Thomas, Qian Chen Yong, Rodolfo M Rosa, et al.
Circulation. Heart Failure|March 6, 2015
Phosphorylation of cardiac Myosin-binding protein-C is a critical mediator of diastolic functionPaola C Rosas, Yang Liu, Mohamed I Abdalla, et al.
Pageof 2