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ACS Chemical Neuroscience|April 24, 2023
Aberrant SENP1-SUMO-Sirt3 Signaling Causes the Disturbances of Mitochondrial Deacetylation and Oxidative Phosphorylation in Prion-Infected Animal and Cell ModelsDong-Dong Chen, Qi Shi, Xin Liu, et al.
Plos One|February 8, 2011
Familial CJD associated PrP mutants within transmembrane region induced Ctm-PrP retention in ER and triggered apoptosis by ER stress in SH-SY5Y cellsXin Wang, Qi Shi, Kun Xu, et al.
Plos One|September 10, 2011
The epidemiological, clinical, and laboratory features of sporadic Creutzfeldt-Jakob disease patients in China: surveillance data from 2006 to 2010Chen Gao, Qi Shi, Chan Tian, et al.
International Journal of Molecular Medicine|May 4, 2011
Knockdown of prion protein (PrP) by RNA interference weakens the protective activity of wild-type PrP against copper ion and antagonizes the cytotoxicity of fCJD-associated PrP mutants in cultured cellsZhao-Yun Wang, Chan Tian, Yuan-Yuan Jing, et al.
Biomedical and Environmental Sciences : BES|September 21, 2022
Global Profiles of Acetylated Proteins in Brains of Scrapie Agents 139A- and ME7-Infected Mice Collected at Mid-Early, Mid-Late, and Terminal StagesQi Shi, Dong Dong Chen, Maimaitiming Adalati, et al.
Prion|December 10, 2019
Significant enhanced expressions of aquaporin-1, -4 and -9 in the brains of various prion diseasesQi Shi, Yue-Zhang Wu, Xuehua Yang, et al.
International Journal of Molecular Medicine|January 15, 2013
Global transcriptional profiling of the postmortem brain of a patient with G114V genetic Creutzfeldt-Jakob diseaseChan Tian, Di Liu, Chen Chen, et al.
Molecular Neurobiology|October 15, 2014
Remarkable Activation of the Complement System and Aberrant Neuronal Localization of the Membrane Attack Complex in the Brain Tissues of Scrapie-Infected RodentsYan Lv, Cao Chen, Bao-Yun Zhang, et al.
Frontiers in Public Health|June 28, 2024
Updated global epidemiology atlas of human prion diseasesLi-Ping Gao, Ting-Ting Tian, Kang Xiao, et al.
BMJ Open|November 16, 2021
Characteristics of Chinese patients with genetic CJD who have E196A or E196K mutation in PRNP: comparative analysis of patients identified in the Chinese National CJD Surveillance SystemQi Shi, Kang Xiao, Cao Chen, et al.
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