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Current Opinion in Hematology|October 24, 2024
PROTACs in platelets: emerging antithrombotic strategies and future perspectivesJustin S Trory, Jordan Vautrinot, Carl J May, et al.Plos One|February 13, 2025
Biomarkers to predict or measure steroid resistance in idiopathic nephrotic syndrome: A systematic reviewCarl J May, Nathan P Ford, Gavin I Welsh, et al.Clinical Kidney Journal|May 20, 2024
Genome editing and kidney healthNadia Tavakolidakhrabadi, Francesco Aulicino, Carl J May, et al.Iscience|August 1, 2026
A Rac-PI3Kβ feedback loop drives GPVI-dependent platelet activationTom N Durrant, James L Hutchinson, Carl J May, et al.Cell Communication and Signaling : CCS|May 14, 2026
PLCε regulates podocyte differentiation and TGF-β1 responses via alteration of SMAD2/SMAD3 ratioCarl J May, Sarah E Hunter, Agnieszka Bierzynska, et al.American Journal of Physiology. Renal Physiology|July 25, 2019
Human Th17 cells produce a soluble mediator that increases podocyte motility via signaling pathways that mimic PAR-1 activationCarl J May, Gavin I Welsh, Musleeha Chesor, et al.Molecular Therapy : the Journal of the American Society of Gene Therapy|October 8, 2025
Podocyte-directed VEGFC gene therapy prevents increased glomerular permeability and glycocalyx damage in experimental type 1 diabetesAldara Martin Alonso, Carl J May, Holly Stowell-Connolly, et al.Kidney International|March 20, 2023
Podocyte protease activated receptor 1 stimulation in mice produces focal segmental glomerulosclerosis mirroring human disease signaling eventsCarl J May, Musleeha Chesor, Sarah E Hunter, et al.Pageof 1