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Carl de Trez

Showing results (21-30 of 53) with videos related to

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Infection and Immunity|April 22, 2003
Amastigote load and cell surface phenotype of infected cells from lesions and lymph nodes of susceptible and resistant mice infected with Leishmania majorEric Muraille, Carl De Trez, Bernard Pajak, et al.
Plos Pathogens|July 22, 2016
The Dermis as a Delivery Site of Trypanosoma brucei for Tsetse FliesGuy Caljon, Nick Van Reet, Carl De Trez, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 9, 2005
TLR4 and Toll-IL-1 receptor domain-containing adapter-inducing IFN-beta, but not MyD88, regulate Escherichia coli-induced dendritic cell maturation and apoptosis in vivoCarl De Trez, Bernard Pajak, Maryse Brait, et al.
Journal of Immunology (Baltimore, Md. : 1950)|April 3, 2016
Identification of Immune Effectors Essential to the Control of Primary and Secondary Intranasal Infection with Brucella melitensis in MiceDelphine Hanot Mambres, Arnaud Machelart, Georges Potemberg, et al.
Plos Pathogens|February 4, 2020
Hepatocyte-derived IL-10 plays a crucial role in attenuating pathogenicity during the chronic phase of T. congolense infectionBenoit Stijlemans, Hannelie Korf, Patrick De Baetselier, et al.
Parasite Immunology|July 21, 2019
Trypanosoma brucei brucei causes a rapid and persistent influx of neutrophils in the spleen of infected miceViolette Deleeuw, Hien Thi Thu Phạm, Isabel De Poorter, et al.
Plos One|September 17, 2015
In Situ Characterization of Splenic Brucella melitensis Reservoir Cells during the Chronic Phase of Infection in Susceptible MiceDelphine Hanot Mambres, Arnaud Machelart, Jean-Marie Vanderwinden, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 21, 2014
Humoral immunity and CD4+ Th1 cells are both necessary for a fully protective immune response upon secondary infection with Brucella melitensisMarie-Alice Vitry, Delphine Hanot Mambres, Carl De Trez, et al.
Journal of Immunology (Baltimore, Md. : 1950)|August 7, 2007
OX40 costimulation promotes persistence of cytomegalovirus-specific CD8 T Cells: A CD4-dependent mechanismIan R Humphreys, Andrea Loewendorf, Carl de Trez, et al.
The Journal of Clinical Investigation|July 28, 2007
SLAT regulates Th1 and Th2 inflammatory responses by controlling Ca2+/NFAT signalingStéphane Bécart, Céline Charvet, Ann J Canonigo Balancio, et al.
Pageof 6

Showing results (21-30 of 53) with videos related to

Sort By:
Pageof 6
Infection and Immunity|April 22, 2003
Amastigote load and cell surface phenotype of infected cells from lesions and lymph nodes of susceptible and resistant mice infected with Leishmania majorEric Muraille, Carl De Trez, Bernard Pajak, et al.
Plos Pathogens|July 22, 2016
The Dermis as a Delivery Site of Trypanosoma brucei for Tsetse FliesGuy Caljon, Nick Van Reet, Carl De Trez, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 9, 2005
TLR4 and Toll-IL-1 receptor domain-containing adapter-inducing IFN-beta, but not MyD88, regulate Escherichia coli-induced dendritic cell maturation and apoptosis in vivoCarl De Trez, Bernard Pajak, Maryse Brait, et al.
Journal of Immunology (Baltimore, Md. : 1950)|April 3, 2016
Identification of Immune Effectors Essential to the Control of Primary and Secondary Intranasal Infection with Brucella melitensis in MiceDelphine Hanot Mambres, Arnaud Machelart, Georges Potemberg, et al.
Plos Pathogens|February 4, 2020
Hepatocyte-derived IL-10 plays a crucial role in attenuating pathogenicity during the chronic phase of T. congolense infectionBenoit Stijlemans, Hannelie Korf, Patrick De Baetselier, et al.
Parasite Immunology|July 21, 2019
Trypanosoma brucei brucei causes a rapid and persistent influx of neutrophils in the spleen of infected miceViolette Deleeuw, Hien Thi Thu Phạm, Isabel De Poorter, et al.
Plos One|September 17, 2015
In Situ Characterization of Splenic Brucella melitensis Reservoir Cells during the Chronic Phase of Infection in Susceptible MiceDelphine Hanot Mambres, Arnaud Machelart, Jean-Marie Vanderwinden, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 21, 2014
Humoral immunity and CD4+ Th1 cells are both necessary for a fully protective immune response upon secondary infection with Brucella melitensisMarie-Alice Vitry, Delphine Hanot Mambres, Carl De Trez, et al.
Journal of Immunology (Baltimore, Md. : 1950)|August 7, 2007
OX40 costimulation promotes persistence of cytomegalovirus-specific CD8 T Cells: A CD4-dependent mechanismIan R Humphreys, Andrea Loewendorf, Carl de Trez, et al.
The Journal of Clinical Investigation|July 28, 2007
SLAT regulates Th1 and Th2 inflammatory responses by controlling Ca2+/NFAT signalingStéphane Bécart, Céline Charvet, Ann J Canonigo Balancio, et al.
Pageof 6