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Proceedings of the National Academy of Sciences of the United States of America|December 5, 2013
PirB regulates a structural substrate for cortical plasticityMaja Djurisic, George S Vidal, Miriam Mann, et al.Neuron|December 1, 2009
Classical MHCI molecules regulate retinogeniculate refinement and limit ocular dominance plasticityAkash Datwani, Michael J McConnell, Patrick O Kanold, et al.Proceedings of the National Academy of Sciences of the United States of America|September 18, 2025
C4d, a high-affinity LilrB2 ligand, is elevated in Alzheimer's disease and mediates synapse pruningBarbara K Brott, Aram J Raissi, Kristina D Micheva, et al.Elife|February 25, 2017
A saturation hypothesis to explain both enhanced and impaired learning with enhanced plasticityTd Barbara Nguyen-Vu, Grace Q Zhao, Subhaneil Lahiri, et al.Science Translational Medicine|October 17, 2014
Blocking PirB up-regulates spines and functional synapses to unlock visual cortical plasticity and facilitate recovery from amblyopiaDavid N Bochner, Richard W Sapp, Jaimie D Adelson, et al.Cerebral Cortex (New York, N.Y. : 1991)|October 16, 2014
Developmental Sculpting of Intracortical Circuits by MHC Class I H2-Db and H2-KbJaimie D Adelson, Richard W Sapp, Barbara K Brott, et al.Science (New York, N.Y.)|September 21, 2013
Human LilrB2 is a β-amyloid receptor and its murine homolog PirB regulates synaptic plasticity in an Alzheimer's modelTaeho Kim, George S Vidal, Maja Djurisic, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 8, 2012
Synaptic plasticity defect following visual deprivation in Alzheimer's disease model transgenic miceChristopher M William, Mark L Andermann, Glenn J Goldey, et al.Neuron|March 27, 2012
Neuroprotection from stroke in the absence of MHCI or PirBJaimie D Adelson, George E Barreto, Lijun Xu, et al.Pageof 3