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Carlo Colombo

Showing results (21-30 of 29) with videos related to

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Nano Letters|August 29, 2012
An electrically-driven GaAs nanowire surface plasmon sourcePengyu Fan, Carlo Colombo, Kevin C Y Huang, et al.
Diabetes Care|October 9, 2008
Insulin gene mutations as cause of diabetes in children negative for five type 1 diabetes autoantibodiesRiccardo Bonfanti, Carlo Colombo, Valentina Nocerino, et al.
The Journal of Biological Chemistry|November 29, 2002
Opposite effects of background genotype on muscle and liver insulin sensitivity of lipoatrophic mice. Role of triglyceride clearanceCarlo Colombo, Martin Haluzik, Jaime J Cutson, et al.
Endocrinology|April 3, 2004
Genetic background (C57BL/6J versus FVB/N) strongly influences the severity of diabetes and insulin resistance in ob/ob miceMartin Haluzik, Carlo Colombo, Oksana Gavrilova, et al.
Pediatric Diabetes|June 15, 2012
Glyburide ameliorates motor coordination and glucose homeostasis in a child with diabetes associated with the KCNJ11/S225T, del226-232 mutationDomenica Battaglia, Yu-Wen Lin, Claudia Brogna, et al.
Diabetes|October 29, 2010
Congenital hyperinsulinism and glucose hypersensitivity in homozygous and heterozygous carriers of Kir6.2 (KCNJ11) mutation V290M mutation: K(ATP) channel inactivation mechanism and clinical managementKaren J Loechner, Alejandro Akrouh, Harley T Kurata, et al.
Diabetes Care|July 26, 2014
No sign of proliferative retinopathy in 15 patients with permanent neonatal diabetes with a median diabetes duration of 24 yearsDario Iafusco, Silvana Salardi, Giovanni Chiari, et al.
The Journal of Clinical Investigation|May 3, 2008
Seven mutations in the human insulin gene linked to permanent neonatal/infancy-onset diabetes mellitusCarlo Colombo, Ottavia Porzio, Ming Liu, et al.
Molecular Endocrinology (Baltimore, Md.)|November 4, 2009
Opposite clinical phenotypes of glucokinase disease: Description of a novel activating mutation and contiguous inactivating mutations in human glucokinase (GCK) geneFabrizio Barbetti, Nadia Cobo-Vuilleumier, Carlo Dionisi-Vici, et al.
Pageof 3

Showing results (21-30 of 29) with videos related to

Sort By:
Pageof 3
You have reached the last page of results.This site can display upto 29 results.
Nano Letters|August 29, 2012
An electrically-driven GaAs nanowire surface plasmon sourcePengyu Fan, Carlo Colombo, Kevin C Y Huang, et al.
Diabetes Care|October 9, 2008
Insulin gene mutations as cause of diabetes in children negative for five type 1 diabetes autoantibodiesRiccardo Bonfanti, Carlo Colombo, Valentina Nocerino, et al.
The Journal of Biological Chemistry|November 29, 2002
Opposite effects of background genotype on muscle and liver insulin sensitivity of lipoatrophic mice. Role of triglyceride clearanceCarlo Colombo, Martin Haluzik, Jaime J Cutson, et al.
Endocrinology|April 3, 2004
Genetic background (C57BL/6J versus FVB/N) strongly influences the severity of diabetes and insulin resistance in ob/ob miceMartin Haluzik, Carlo Colombo, Oksana Gavrilova, et al.
Pediatric Diabetes|June 15, 2012
Glyburide ameliorates motor coordination and glucose homeostasis in a child with diabetes associated with the KCNJ11/S225T, del226-232 mutationDomenica Battaglia, Yu-Wen Lin, Claudia Brogna, et al.
Diabetes|October 29, 2010
Congenital hyperinsulinism and glucose hypersensitivity in homozygous and heterozygous carriers of Kir6.2 (KCNJ11) mutation V290M mutation: K(ATP) channel inactivation mechanism and clinical managementKaren J Loechner, Alejandro Akrouh, Harley T Kurata, et al.
Diabetes Care|July 26, 2014
No sign of proliferative retinopathy in 15 patients with permanent neonatal diabetes with a median diabetes duration of 24 yearsDario Iafusco, Silvana Salardi, Giovanni Chiari, et al.
The Journal of Clinical Investigation|May 3, 2008
Seven mutations in the human insulin gene linked to permanent neonatal/infancy-onset diabetes mellitusCarlo Colombo, Ottavia Porzio, Ming Liu, et al.
Molecular Endocrinology (Baltimore, Md.)|November 4, 2009
Opposite clinical phenotypes of glucokinase disease: Description of a novel activating mutation and contiguous inactivating mutations in human glucokinase (GCK) geneFabrizio Barbetti, Nadia Cobo-Vuilleumier, Carlo Dionisi-Vici, et al.
Pageof 3