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Biological Psychiatry|September 3, 2016
CRTC1 Function During Memory Encoding Is Disrupted in NeurodegenerationArnaldo Parra-Damas, Meng Chen, Lilian Enriquez-Barreto, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 17, 2023
Activity-Dependent Nr4a2 Induction Modulates Synaptic Expression of AMPA Receptors and Plasticity via a Ca<sup>2+/</sup>CRTC1/CREB PathwayJudit Català-Solsona, Pablo J Lituma, Stefano Lutzu, et al.
Scientific Reports|April 26, 2018
Reversal of memory and neuropsychiatric symptoms and reduced tau pathology by selenium in 3xTg-AD miceAnn Van der Jeugd, Arnaldo Parra-Damas, Raquel Baeta-Corral, et al.
Eneuro|March 25, 2020
Proteasomal-Mediated Degradation of AKAP150 Accompanies AMPAR Endocytosis during cLTDWenwen Cheng, Dolores Siedlecki-Wullich, Judit Català-Solsona, et al.
ACS Pharmacology & Translational Science|September 19, 2025
Cannabinoid CB<sub>1</sub> Receptor Activation Mitigates <i>N</i>‑Methyl‑d‑aspartate Receptor-Mediated NeurotoxicityGemma Navarro, Iu Raïch, Joan Biel Rebassa, et al.
Molecular Psychiatry|January 30, 2025
Synaptic vulnerability to amyloid-β and tau pathologies differentially disrupts emotional and memory neural circuitsMaria Dolores Capilla-López, Angel Deprada, Yuniesky Andrade-Talavera, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|December 13, 2017
Proteolytic Processing of Neurexins by Presenilins Sustains Synaptic Vesicle ReleaseEmilia Servián-Morilla, Estefanía Robles-Lanuza, Ana C Sánchez-Hidalgo, et al.
Biorxiv : the Preprint Server for Biology|February 8, 2024
Nr4a2 blocks oAβ-mediated synaptic plasticity dysfunction and ameliorates spatial memory deficits in the APP <sub>Sw,Ind</sub> mouseJudit Català-Solsona, Stefano Lutzu, Pablo J Lituma, et al.
Neurobiology of Aging|August 8, 2016
Preservation of cell-survival mechanisms by the presenilin-1 K239N mutation may cause its milder clinical phenotypeSara Sarroca, Patricia Molina-Martínez, Cristina Aresté, et al.
Neuron|April 7, 2004
Loss of presenilin function causes impairments of memory and synaptic plasticity followed by age-dependent neurodegenerationCarlos A Saura, Se-Young Choi, Vassilios Beglopoulos, et al.
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