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The Journal of Physiology|March 8, 2002
Functional characterization of recombinant human ClC-4 chloride channels in cultured mammalian cellsCarlos G Vanoye, Alfred L George
Molecular Pharmacology|May 4, 2016
Use-Dependent Block of Human Cardiac Sodium Channels by GS967Franck Potet, Carlos G Vanoye, Alfred L George
Current Opinion in Nephrology and Hypertension|August 18, 2006
Molecular physiology of renal ClC chloride channels/transportersSaba Sile, Carlos G Vanoye, Alfred L George
Channels (Austin, Tex.)|August 12, 2008
Exaggerated Mg2+ inhibition of Kir2.1 as a consequence of reduced PIP2 sensitivity in Andersen syndromeLeomar Y Ballester, Carlos G Vanoye, Alfred L George
The Journal of General Physiology|December 29, 2005
Single-channel properties of human NaV1.1 and mechanism of channel dysfunction in SCN1A-associated epilepsyCarlos G Vanoye, Christoph Lossin, Thomas H Rhodes, et al.
The Journal of Clinical Investigation|December 19, 2002
Functional repair of a mutant chloride channel using a trans-splicing ribozymeChristopher S Rogers, Carlos G Vanoye, Bruce A Sullenger, et al.
The Journal of Physiology|November 26, 2008
KCNE4 domains required for inhibition of KCNQ1Lauren J Manderfield, Melissa A Daniels, Carlos G Vanoye, et al.
The Journal of Biological Chemistry|December 2, 2010
KCNE4 juxtamembrane region is required for interaction with calmodulin and for functional suppression of KCNQ1Erin J Ciampa, Richard C Welch, Carlos G Vanoye, et al.
The Journal of General Physiology|January 30, 2013
Mechanism of sodium channel NaV1.9 potentiation by G-protein signalingCarlos G Vanoye, Jennifer D Kunic, George R Ehring, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 21, 2004
Noninactivating voltage-gated sodium channels in severe myoclonic epilepsy of infancyThomas H Rhodes, Christoph Lossin, Carlos G Vanoye, et al.
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