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Carol F Webb

Showing results (21-30 of 33) with videos related to

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Arthritis & Rheumatology (Hoboken, N.J.)|September 5, 2014
Disease activity in systemic lupus erythematosus correlates with expression of the transcription factor AT-rich-interactive domain 3AJulie M Ward, Kira Rose, Courtney Montgomery, et al.
Stem Cells (Dayton, Ohio)|August 4, 2010
Loss of Bright/ARID3a function promotes developmental plasticityGuangyu An, Cathrine A Miner, Jamee C Nixon, et al.
Data in Brief|September 23, 2016
Expression and methylation data from SLE patient and healthy control blood samples subdivided with respect to ARID3a levelsJulie M Ward, Michelle L Ratliff, Mikhail G Dozmorov, et al.
Frontiers in Immunology|January 31, 2019
Insights From Analysis of Human Antigen-Specific Memory B Cell RepertoiresHemangi B Shah, Kenneth Smith, Jonathan D Wren, et al.
Journal of Autoimmunity|August 15, 2016
Human effector B lymphocytes express ARID3a and secrete interferon alphaJulie M Ward, Michelle L Ratliff, Mikhail G Dozmorov, et al.
Frontiers in Immunology|June 6, 2019
Mechanistic Characterization of <i>RASGRP1</i> Variants Identifies an hnRNP-K-Regulated Transcriptional Enhancer Contributing to SLE SusceptibilityJulio E Molineros, Bhupinder Singh, Chikashi Terao, et al.
Molecular and Cellular Biology|January 5, 2011
The ARID family transcription factor bright is required for both hematopoietic stem cell and B lineage developmentCarol F Webb, James Bryant, Melissa Popowski, et al.
Stem Cell Reports|February 11, 2014
Bright/Arid3A acts as a barrier to somatic cell reprogramming through direct regulation of Oct4, Sox2, and NanogMelissa Popowski, Troy D Templeton, Bum-Kyu Lee, et al.
Journal of Autoimmunity|October 10, 2018
ARID3a gene profiles are strongly associated with human interferon alpha productionMichelle L Ratliff, Joshua Garton, Lori Garman, et al.
The EMBO Journal|February 14, 2009
Signalling of the BCR is regulated by a lipid rafts-localised transcription factor, BrightChristian Schmidt, Dongkyoon Kim, Gregory C Ippolito, et al.
Pageof 4

Showing results (21-30 of 33) with videos related to

Sort By:
Pageof 4
Arthritis & Rheumatology (Hoboken, N.J.)|September 5, 2014
Disease activity in systemic lupus erythematosus correlates with expression of the transcription factor AT-rich-interactive domain 3AJulie M Ward, Kira Rose, Courtney Montgomery, et al.
Stem Cells (Dayton, Ohio)|August 4, 2010
Loss of Bright/ARID3a function promotes developmental plasticityGuangyu An, Cathrine A Miner, Jamee C Nixon, et al.
Data in Brief|September 23, 2016
Expression and methylation data from SLE patient and healthy control blood samples subdivided with respect to ARID3a levelsJulie M Ward, Michelle L Ratliff, Mikhail G Dozmorov, et al.
Frontiers in Immunology|January 31, 2019
Insights From Analysis of Human Antigen-Specific Memory B Cell RepertoiresHemangi B Shah, Kenneth Smith, Jonathan D Wren, et al.
Journal of Autoimmunity|August 15, 2016
Human effector B lymphocytes express ARID3a and secrete interferon alphaJulie M Ward, Michelle L Ratliff, Mikhail G Dozmorov, et al.
Frontiers in Immunology|June 6, 2019
Mechanistic Characterization of <i>RASGRP1</i> Variants Identifies an hnRNP-K-Regulated Transcriptional Enhancer Contributing to SLE SusceptibilityJulio E Molineros, Bhupinder Singh, Chikashi Terao, et al.
Molecular and Cellular Biology|January 5, 2011
The ARID family transcription factor bright is required for both hematopoietic stem cell and B lineage developmentCarol F Webb, James Bryant, Melissa Popowski, et al.
Stem Cell Reports|February 11, 2014
Bright/Arid3A acts as a barrier to somatic cell reprogramming through direct regulation of Oct4, Sox2, and NanogMelissa Popowski, Troy D Templeton, Bum-Kyu Lee, et al.
Journal of Autoimmunity|October 10, 2018
ARID3a gene profiles are strongly associated with human interferon alpha productionMichelle L Ratliff, Joshua Garton, Lori Garman, et al.
The EMBO Journal|February 14, 2009
Signalling of the BCR is regulated by a lipid rafts-localised transcription factor, BrightChristian Schmidt, Dongkyoon Kim, Gregory C Ippolito, et al.
Pageof 4