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The Journal of Biological Chemistry|November 22, 2017
Nitro-fatty acid inhibition of triple-negative breast cancer cell viability, migration, invasion, and tumor growthChen-Shan Chen Woodcock, Yi Huang, Steven R Woodcock, et al.Nature|August 2, 2003
Essential role for the peroxiredoxin Prdx1 in erythrocyte antioxidant defence and tumour suppressionCarola A Neumann, Daniela S Krause, Christopher V Carman, et al.The Journal of Biological Chemistry|November 28, 2018
Electrophilic fatty acids impair RAD51 function and potentiate the effects of DNA-damaging agents on growth of triple-negative breast cellsAlparslan Asan, John J Skoko, Chen-Shan Chen Woodcock, et al.Cancer Letters|April 13, 2026
Nitroalkenes Exploit Dependence on Autophagy-Lysosome Pathway in PARPi-Resistant Triple Negative Breast CancerLisa Hong, Sanghoon Lee, Leonard Frisbie, et al.Research Square|April 8, 2025
Chemoproteomic analysis reveals RECQL4 as a mediator of nitroalkene-dependent double-strand break repair inhibition in cancerDennis C Braden, Mostafa A L Adbel-Salam, Alparslan Asan, et al.Antioxidants & Redox Signaling|April 12, 2017
A Peroxidase Peroxiredoxin 1-Specific Redox Regulation of the Novel FOXO3 microRNA Target let-7Barbara L Hopkins, Monica Nadler, John J Skoko, et al.Redox Biology|September 4, 2022
Redox regulation of RAD51 Cys319 and homologous recombination by peroxiredoxin 1John J Skoko, Juxiang Cao, David Gaboriau, et al.Redox Biology|August 26, 2023
Small molecule nitroalkenes inhibit RAD51-mediated homologous recombination and amplify triple-negative breast cancer cell killing by DNA-directed therapiesLisa Hong, Dennis C Braden, Yaoning Zhao, et al.Biorxiv : the Preprint Server for Biology|August 30, 2023
Small molecule nitroalkenes inhibit RAD51-mediated homologous recombination and amplify triple-negative breast cancer cell killing by DNA-directed therapiesLisa Hong, Dennis C Braden, Yaoning Zhao, et al.Pageof 3