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Frontiers in Aging Neuroscience|October 29, 2020
Clinically Precedented Protein Kinases: Rationale for Their Use in Neurodegenerative DiseaseCaroline L Benn, Lee A Dawson
Molecular Neurodegeneration|October 29, 2008
Optimisation of region-specific reference gene selection and relative gene expression analysis methods for pre-clinical trials of Huntington's diseaseCaroline L Benn, Helen Fox, Gillian P Bates
Journal of Huntington'S Disease|September 14, 2020
Drugging DNA Damage Repair Pathways for Trinucleotide Repeat Expansion DiseasesCaroline L Benn, Karl R Gibson, David S Reynolds
Methods in Molecular Biology (Clifton, N.J.)|June 18, 2004
Neurotransmitter receptor analysis in transgenic mouse modelsCaroline L Benn, Laurie A Farrell, Jang-Ho J Cha
Journal of Cellular and Molecular Medicine|July 16, 2009
The polyubiquitin Ubc gene modulates histone H2A monoubiquitylation in the R6/2 mouse model of Huntington's diseaseJohn S Bett, Caroline L Benn, Kwon-Yul Ryu, et al.
Frontiers in Medicine|June 16, 2018
Physiology of Hyperuricemia and Urate-Lowering TreatmentsCaroline L Benn, Pinky Dua, Rachel Gurrell, et al.
Plos One|June 2, 2009
Genetic knock-down of HDAC7 does not ameliorate disease pathogenesis in the R6/2 mouse model of Huntington's diseaseCaroline L Benn, Rachel Butler, Lydia Mariner, et al.
Plos One|December 6, 2011
SAHA decreases HDAC 2 and 4 levels in vivo and improves molecular phenotypes in the R6/2 mouse model of Huntington's diseaseMichal Mielcarek, Caroline L Benn, Sophie A Franklin, et al.
Stem Cells Translational Medicine|April 27, 2016
Targeting the cAMP and Transforming Growth Factor-β Pathway Increases Proliferation to Promote Re-Epithelialization of Human Stem Cell-Derived Retinal Pigment EpitheliumParul Choudhary, Alex Gutteridge, Emma Impey, et al.
Scientific Reports|September 21, 2017
Disruption to schizophrenia-associated gene Fez1 in the hippocampus of HDAC11 knockout miceDale T Bryant, Christian Landles, Aikaterini S Papadopoulou, et al.
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