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American Journal of Physiology. Cell Physiology|May 1, 2023
Modulation of cytoskeleton in cardiomyopathy caused by mutations in <i>LMNA</i> geneMaria Chatzifrangkeskou, Caroline Le Dour, Antoine MuchirThe International Journal of Biochemistry & Cell Biology|March 12, 2011
Molecular mechanisms of human lipodystrophies: from adipocyte lipid droplet to oxidative stress and lipotoxicityCorinne Vigouroux, Martine Caron-Debarle, Caroline Le Dour, et al.Journal of Lipid Research|November 16, 2016
Extracellular matrix remodeling and transforming growth factor-β signaling abnormalities induced by lamin A/C variants that cause lipodystrophyCaroline Le Dour, Wei Wu, Véronique Béréziat, et al.The American Journal of Pathology|September 28, 2011
LMNA mutations induce a non-inflammatory fibrosis and a brown fat-like dystrophy of enlarged cervical adipose tissueVéronique Béréziat, Pascale Cervera, Caroline Le Dour, et al.Human Molecular Genetics|January 11, 2017
Decreased WNT/β-catenin signalling contributes to the pathogenesis of dilated cardiomyopathy caused by mutations in the lamin a/C geneCaroline Le Dour, Coline Macquart, Fusako Sera, et al.Human Molecular Genetics|June 13, 2018
Microtubule cytoskeleton regulates Connexin 43 localization and cardiac conduction in cardiomyopathy caused by mutation in A-type lamins geneColine Macquart, Rene Jüttner, Blanca Morales Rodriguez, et al.Nucleus (Austin, Tex.)|May 27, 2014
Depletion of lamina-associated polypeptide 1 from cardiomyocytes causes cardiac dysfunction in miceJi-Yeon Shin, Caroline Le Dour, Fusako Sera, et al.Nucleus (Austin, Tex.)|March 27, 2018
Lipodystrophic syndromes due to LMNA mutations: recent developments on biomolecular aspects, pathophysiological hypotheses and therapeutic perspectivesCorinne Vigouroux, Anne-Claire Guénantin, Camille Vatier, et al.The Journal of Clinical Endocrinology and Metabolism|February 25, 2011
A homozygous mutation of prelamin-A preventing its farnesylation and maturation leads to a severe lipodystrophic phenotype: new insights into the pathogenicity of nonfarnesylated prelamin-ACaroline Le Dour, Stéphane Schneebeli, Fawzi Bakiri, et al.Human Molecular Genetics|May 1, 2016
ERK1/2 directly acts on CTGF/CCN2 expression to mediate myocardial fibrosis in cardiomyopathy caused by mutations in the lamin A/C geneMaria Chatzifrangkeskou, Caroline Le Dour, Wei Wu, et al.Pageof 2