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Cells|December 24, 2025
Hyperactivity and Differential Gene Expression in <i>lbx1a<sup>(</sup><sup>-/</sup><sup>-)</sup></i> Zebrafish LarvaeCarsten Drepper, Laura Kettenstock, Simon Stöckl, et al.European Child & Adolescent Psychiatry|December 23, 2019
Anxiety risk SNPs on chromosome 2 modulate arousal in children in a fear generalization paradigmJulia Reinhard, Carsten Drepper, Heike Weber, et al.Nature Genetics|October 26, 2005
Mutation of Vps54 causes motor neuron disease and defective spermiogenesis in the wobbler mouseThomas Schmitt-John, Carsten Drepper, Anke Mussmann, et al.Nucleic Acids Research|December 1, 2021
Loss of full-length hnRNP R isoform impairs DNA damage response in motoneurons by inhibiting Yb1 recruitment to chromatinHanaa Ghanawi, Luisa Hennlein, Abdolhossein Zare, et al.Nature Neuroscience|November 8, 2016
C9ORF72 interaction with cofilin modulates actin dynamics in motor neuronsRajeeve Sivadasan, Daniel Hornburg, Carsten Drepper, et al.Acta Neuropathologica|May 31, 2015
Dysregulated IGFBP5 expression causes axon degeneration and motoneuron loss in diabetic neuropathyChristian M Simon, Stefanie Rauskolb, Jennifer M Gunnersen, et al.Nature Cell Biology|May 26, 2009
A functional screen implicates microRNA-138-dependent regulation of the depalmitoylation enzyme APT1 in dendritic spine morphogenesisGabriele Siegel, Gregor Obernosterer, Roberto Fiore, et al.Brain : a Journal of Neurology|February 28, 2012
Clinical characteristics of patients with familial amyotrophic lateral sclerosis carrying the pathogenic GGGGCC hexanucleotide repeat expansion of C9ORF72Adriano Chiò, Giuseppe Borghero, Gabriella Restagno, et al.Nature Neuroscience|April 2, 2014
Mutations in the Matrin 3 gene cause familial amyotrophic lateral sclerosisJanel O Johnson, Erik P Pioro, Ashley Boehringer, et al.The Lancet. Neurology|March 13, 2012
Frequency of the C9orf72 hexanucleotide repeat expansion in patients with amyotrophic lateral sclerosis and frontotemporal dementia: a cross-sectional studyElisa Majounie, Alan E Renton, Kin Mok, et al.Pageof 3