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Caterina Bendotti

Showing results (41-50 of 104) with videos related to

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Brain Pathology (Zurich, Switzerland)|August 4, 2019
P2X7 activation enhances skeletal muscle metabolism and regeneration in SOD1G93A mouse model of amyotrophic lateral sclerosisPaola Fabbrizio, Savina Apolloni, Andrea Bianchi, et al.
Experimental Neurology|October 24, 2025
Beneficial effects of synthetic torpor in a fast-progressing mouse model of amyotrophic lateral sclerosisStefano Fabrizio Columbro, Massimo Tortarolo, Andrea David Re Cecconi, et al.
Biochemical and Biophysical Research Communications|January 2, 2007
Proteomic analysis of spinal cord of presymptomatic amyotrophic lateral sclerosis G93A SOD1 mouseTania Massignan, Filippo Casoni, Manuela Basso, et al.
The European Journal of Neuroscience|August 15, 2006
Cell culture models to investigate the selective vulnerability of motoneuronal mitochondria to familial ALS-linked G93ASOD1Andrea Raimondi, Alessandra Mangolini, Milena Rizzardini, et al.
Antioxidants & Redox Signaling|May 9, 2012
Molecular signatures of amyotrophic lateral sclerosis disease progression in hind and forelimb muscles of an SOD1(G93A) mouse modelDaniele Capitanio, Michele Vasso, Antonia Ratti, et al.
Pharmacological Research|December 8, 2015
Synthetic and natural small molecule TLR4 antagonists inhibit motoneuron death in cultures from ALS mouse modelMassimiliano De Paola, Stefania E Sestito, Alessandro Mariani, et al.
Plos One|February 6, 2014
Increased axonal ribosome numbers is an early event in the pathogenesis of amyotrophic lateral sclerosisMark H G Verheijen, Marco Peviani, Rita Hendricusdottir, et al.
Human Molecular Genetics|February 25, 2016
ALS mouse model SOD1G93A displays early pathology of sensory small fibers associated to accumulation of a neurotoxic splice variant of peripherinJenny Sassone, Michela Taiana, Raffaella Lombardi, et al.
Plos One|August 13, 2013
A mouse model of familial ALS has increased CNS levels of endogenous ubiquinol9/10 and does not benefit from exogenous administration of ubiquinol10Jacopo Lucchetti, Marianna Marino, Simonetta Papa, et al.
Cytokine|December 31, 2003
Glial activation and TNFR-I upregulation precedes motor dysfunction in the spinal cord of mnd miceTiziana Mennini, Paolo Bigini, Alfredo Cagnotto, et al.
Pageof 11

Showing results (41-50 of 104) with videos related to

Sort By:
Pageof 11
Brain Pathology (Zurich, Switzerland)|August 4, 2019
P2X7 activation enhances skeletal muscle metabolism and regeneration in SOD1G93A mouse model of amyotrophic lateral sclerosisPaola Fabbrizio, Savina Apolloni, Andrea Bianchi, et al.
Experimental Neurology|October 24, 2025
Beneficial effects of synthetic torpor in a fast-progressing mouse model of amyotrophic lateral sclerosisStefano Fabrizio Columbro, Massimo Tortarolo, Andrea David Re Cecconi, et al.
Biochemical and Biophysical Research Communications|January 2, 2007
Proteomic analysis of spinal cord of presymptomatic amyotrophic lateral sclerosis G93A SOD1 mouseTania Massignan, Filippo Casoni, Manuela Basso, et al.
The European Journal of Neuroscience|August 15, 2006
Cell culture models to investigate the selective vulnerability of motoneuronal mitochondria to familial ALS-linked G93ASOD1Andrea Raimondi, Alessandra Mangolini, Milena Rizzardini, et al.
Antioxidants & Redox Signaling|May 9, 2012
Molecular signatures of amyotrophic lateral sclerosis disease progression in hind and forelimb muscles of an SOD1(G93A) mouse modelDaniele Capitanio, Michele Vasso, Antonia Ratti, et al.
Pharmacological Research|December 8, 2015
Synthetic and natural small molecule TLR4 antagonists inhibit motoneuron death in cultures from ALS mouse modelMassimiliano De Paola, Stefania E Sestito, Alessandro Mariani, et al.
Plos One|February 6, 2014
Increased axonal ribosome numbers is an early event in the pathogenesis of amyotrophic lateral sclerosisMark H G Verheijen, Marco Peviani, Rita Hendricusdottir, et al.
Human Molecular Genetics|February 25, 2016
ALS mouse model SOD1G93A displays early pathology of sensory small fibers associated to accumulation of a neurotoxic splice variant of peripherinJenny Sassone, Michela Taiana, Raffaella Lombardi, et al.
Plos One|August 13, 2013
A mouse model of familial ALS has increased CNS levels of endogenous ubiquinol9/10 and does not benefit from exogenous administration of ubiquinol10Jacopo Lucchetti, Marianna Marino, Simonetta Papa, et al.
Cytokine|December 31, 2003
Glial activation and TNFR-I upregulation precedes motor dysfunction in the spinal cord of mnd miceTiziana Mennini, Paolo Bigini, Alfredo Cagnotto, et al.
Pageof 11