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Brain Stimulation|June 3, 2020
LTP-like cortical plasticity predicts conversion to dementia in patients with memory impairmentFrancesco Di Lorenzo, Caterina Motta, Elias Paolo Casula, et al.Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|December 16, 2025
Reframing TDP-43 in Alzheimer's disease: From co-pathology to neurovascular culpritMartina Gaia Di Donna, Caterina Motta, Chiara Giuseppina Bonomi, et al.Journal of Alzheimer'S Disease : JAD|October 30, 2018
Impaired Spike Timing Dependent Cortico-Cortical Plasticity in Alzheimer's Disease PatientsFrancesco Di Lorenzo, Viviana Ponzo, Caterina Motta, et al.European Journal of Neurology|January 24, 2023
Interplay between the catecholaminergic enzymatic axis and neurodegeneration/neuroinflammation processes in the Alzheimer's disease continuumCaterina Motta, Martina Assogna, Chiara Giuseppina Bonomi, et al.European Journal of Neurology|October 12, 2023
Blood-brain barrier permeability is associated with different neuroinflammatory profiles in Alzheimer's diseaseMatilde Bruno, Chiara Giuseppina Bonomi, Francesco Ricci, et al.Minerva Medica|March 12, 2021
Cognitive reserve and Alzheimer's biological continuum: clues for prediction and prevention of dementiaAlessandro Martorana, Martina Assogna, Vincenzo DE Lucia, et al.Biomedicines|March 29, 2023
Functional Correlates of Microglial and Astrocytic Activity in Symptomatic Sporadic Alzheimer's Disease: A CSF/<sup>18</sup>F-FDG-PET StudyChiara Giuseppina Bonomi, Agostino Chiaravalloti, Riccardo Camedda, et al.Journal of Alzheimer'S Disease Reports|April 1, 2025
Cerebrospinal fluid cytokine levels affect electroencephalographic activity in Alzheimer's diseaseMatilde Bruno, Chiara Giuseppina Bonomi, Alessandro Castelli, et al.Neurology|March 20, 2025
Autonomic Dysfunction in Patients With Dementia With Lewy Bodies and Its Relationship With Nigrostriatal DenervationChiara Giuseppina Bonomi, Alessandro Martorana, Caterina Motta, et al.Alzheimer'S Research & Therapy|August 30, 2023
Different associations between amyloid-βeta 42, amyloid-βeta 40, and amyloid-βeta 42/40 with soluble phosphorylated-tau and disease burden in Alzheimer's disease: a cerebrospinal fluid and fluorodeoxyglucose-positron emission tomography studyCaterina Motta, Martina Gaia Di Donna, Chiara Giuseppina Bonomi, et al.Pageof 9