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Gastroenterology Research and Practice|January 2, 2019
How the BRAF V600E Mutation Defines a Distinct Subgroup of Colorectal Cancer: Molecular and Clinical ImplicationsCatherine E Bond, Vicki L J WhitehallCancers|February 11, 2023
The Efficacy of Using Patient-Derived Organoids to Predict Treatment Response in Colorectal CancerChang Su, Kelly A Olsen, Catherine E Bond, et al.Plos One|October 31, 2012
Chromosomal instability in BRAF mutant, microsatellite stable colorectal cancersCatherine E Bond, Aarti Umapathy, Ron L Buttenshaw, et al.BMC Cancer|January 24, 2015
Methylation and expression of the tumour suppressor, PRDM5, in colorectal cancer and polyp subgroupsCatherine E Bond, Mark L Bettington, Sally-Ann Pearson, et al.Plos One|March 22, 2014
Microsatellite stable colorectal cancers stratified by the BRAF V600E mutation show distinct patterns of chromosomal instabilityCatherine E Bond, Derek J Nancarrow, Leesa F Wockner, et al.Cell Reports Methods|December 1, 2022
Comparative analysis of Illumina Mouse Methylation BeadChip and reduced-representation bisulfite sequencing for routine DNA methylation analysisLochlan J Fennell, Gunter Hartel, Diane M McKeone, et al.British Journal of Cancer|March 30, 2021
Aspirin reduces the incidence of metastasis in a pre-clinical study of Braf mutant serrated colorectal neoplasiaAlexandra M Kane, Cheng Liu, Lochlan J Fennell, et al.Oncotarget|September 24, 2016
RNF43 and ZNRF3 are commonly altered in serrated pathway colorectal tumorigenesisCatherine E Bond, Diane M McKeone, Murugan Kalimutho, et al.International Journal of Cancer|May 11, 2011
p53 mutation is common in microsatellite stable, BRAF mutant colorectal cancersCatherine E Bond, Aarti Umapathy, Ingunn Ramsnes, et al.Neoplasia (New York, N.Y.)|January 15, 2020
Alterations in signaling pathways that accompany spontaneous transition to malignancy in a mouse model of BRAF mutant microsatellite stable colorectal cancerAlexandra M Kane, Lochlan J Fennell, Cheng Liu, et al.Pageof 2