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The Journal of Biological Chemistry|April 3, 2002
A highly effective dominant negative alpha s construct containing mutations that affect distinct functions inhibits multiple Gs-coupled receptor signaling pathwaysCatherine H Berlot
Current Opinion in Cell Biology|June 16, 2004
Translating G protein subunit diversity into functional specificityJanet D Robishaw, Catherine H Berlot
Methods in Molecular Biology (Clifton, N.J.)|September 23, 2003
Cellular localization of GFP-tagged alpha subunitsThomas R Hynes, Thomas E Hughes, Catherine H Berlot
Methods (San Diego, Calif.)|July 1, 2008
Multicolor BiFC analysis of competition among G protein beta and gamma subunit interactionsThomas R Hynes, Evan Yost, Stacy Mervine, et al.
Molecular Pharmacology|September 14, 2004
Using molecular tools to dissect the role of Galphas in sensitization of AC1Timothy A Vortherms, Chau H Nguyen, Catherine H Berlot, et al.
Methods in Molecular Biology (Clifton, N.J.)|August 27, 2011
Multicolor BiFC analysis of G protein βγ complex formation and localizationThomas R Hynes, Evan A Yost, Stacy M Yost, et al.
Molecular Pharmacology|June 29, 2007
Live cell analysis of G protein beta5 complex formation, function, and targetingEvan A Yost, Stacy M Mervine, Jonathan L Sabo, et al.
Journal of Molecular Signaling|April 21, 2016
Inhibition of G-Protein βγ Signaling Decreases Levels of Messenger RNAs Encoding Proinflammatory Cytokines in T Cell Receptor-Stimulated CD4(+) T Helper CellsThomas R Hynes, Evan A Yost, Cassandra M Hartle, et al.
The Journal of Biological Chemistry|August 7, 2004
Live cell imaging of Gs and the beta2-adrenergic receptor demonstrates that both alphas and beta1gamma7 internalize upon stimulation and exhibit similar trafficking patterns that differ from that of the beta2-adrenergic receptorThomas R Hynes, Stacy M Mervine, Evan A Yost, et al.
Plos One|January 29, 2015
Inhibition of G-protein βγ signaling enhances T cell receptor-stimulated interleukin 2 transcription in CD4+ T helper cellsEvan A Yost, Thomas R Hynes, Cassandra M Hartle, et al.
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