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Clinical Diabetes and Endocrinology|April 24, 2024
Factors associated with high costs of patients with metabolic dysfunction-associated steatotic liver disease: an observational study using the French CONSTANCES cohortArnaud Nze Ossima, Angélique Brzustowski, Valérie Paradis, et al.Ultrasound in Medicine & Biology|July 13, 2025
Multiparametric ultrasound evaluation of metabolic dysfunction-associated fatty liver disease in minipigsGeorges Chabouh, Basile Pradier, Louise Denis, et al.Nature Communications|May 31, 2018
The histone demethylase Phf2 acts as a molecular checkpoint to prevent NAFLD progression during obesityJulien Bricambert, Marie-Clotilde Alves-Guerra, Pauline Esteves, et al.Hepatology (Baltimore, Md.)|December 17, 2016
Growth factor receptor binding protein 14 inhibition triggers insulin-induced mouse hepatocyte proliferation and is associated with hepatocellular carcinomaLucille Morzyglod, Michèle Caüzac, Lucie Popineau, et al.Molecular Metabolism|June 11, 2025
Deletion of PPARα in mouse brown adipocytes increases their De Novo LipogenesisPierre-Louis Batrow, Sylvie Caspar-Bauguil, Nathalie Rochet, et al.JCI Insight|January 18, 2022
ChREBPβ is dispensable for the control of glucose homeostasis and energy balanceEmeline Recazens, Geneviève Tavernier, Jérémy Dufau, et al.Journal of Hepatology|January 22, 2013
Essential fatty acids deficiency promotes lipogenic gene expression and hepatic steatosis through the liver X receptorSimon Ducheix, Alexandra Montagner, Arnaud Polizzi, et al.Gut|February 4, 2016
Liver PPARα is crucial for whole-body fatty acid homeostasis and is protective against NAFLDAlexandra Montagner, Arnaud Polizzi, Edwin Fouché, et al.Scientific Reports|April 18, 2020
Hepatocyte-specific deletion of Pparα promotes NAFLD in the context of obesityMarion Régnier, Arnaud Polizzi, Sarra Smati, et al.Cell Reports|October 12, 2017
A Specific ChREBP and PPARα Cross-Talk Is Required for the Glucose-Mediated FGF21 ResponseAlison Iroz, Alexandra Montagner, Fadila Benhamed, et al.Pageof 9