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Circulation. Heart Failure|June 4, 2010
Chromogranin B in heart failure: a putative cardiac biomarker expressed in the failing myocardiumHelge Røsjø, Cathrine Husberg, Mai Britt Dahl, et al.Proteomics|July 30, 2017
Mapping the in vitro interactome of cardiac sodium (Na+ )-calcium (Ca2+ ) exchanger 1 (NCX1)Tandekile Lubelwana Hafver, Pimthanya Wanichawan, Ornella Manfra, et al.Cardiovascular Research|April 6, 2019
Coupling of the Na+/K+-ATPase to Ankyrin B controls Na+/Ca2+ exchanger activity in cardiomyocytesJonas Skogestad, Jan Magnus Aronsen, Nils Tovsrud, et al.Frontiers in Pharmacology|June 24, 2021
Design of a Proteolytically Stable Sodium-Calcium Exchanger 1 Activator Peptide for In Vivo StudiesPimthanya Wanichawan, Jonas Skogestad, Marianne Lunde, et al.EMBO Reports|September 29, 2007
AKAP complex regulates Ca2+ re-uptake into heart sarcoplasmic reticulumBirgitte Lygren, Cathrine Rein Carlson, Katja Santamaria, et al.Frontiers in Physiology|January 7, 2025
Wooden breast myopathy is characterized by satellite cell dysfunction and syndecan-4 sheddingLucie Pejšková, Addolorata Pisconti, Marianne Lunde, et al.Frontiers in Cell and Developmental Biology|September 22, 2025
Syndecan-4 interacts directly with β-parvin and regulates the ILK-PINCH-β-parvin complex, the β-parvin-β-PIX-Rac1 axis, and cardiomyocyte geometry in a sex-dependent mannerSabrina Bech Mathiesen, Thea Parsberg Støle, Andreas Romaine, et al.Frontiers in Cell and Developmental Biology|August 28, 2020
Syndecan-4-/- Mice Have Smaller Muscle Fibers, Increased Akt/mTOR/S6K1 and Notch/HES-1 Pathways, and Alterations in Extracellular Matrix ComponentsSissel Beate Rønning, Cathrine Rein Carlson, Jan Magnus Aronsen, et al.The Journal of Biological Chemistry|April 11, 2019
The cardiac syndecan-4 interactome reveals a role for syndecan-4 in nuclear translocation of muscle LIM protein (MLP)Sabrina Bech Mathiesen, Marianne Lunde, Jan Magnus Aronsen, et al.Frontiers in Cell and Developmental Biology|September 12, 2022
The female syndecan-4-/- heart has smaller cardiomyocytes, augmented insulin/pSer473-Akt/pSer9-GSK-3β signaling, and lowered SCOP, pThr308-Akt/Akt and GLUT4 levelsThea Parsberg Støle, Marianne Lunde, Xin Shen, et al.Pageof 3