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Molecular and Cellular Biochemistry|March 16, 2011
Ataxia telangiectasia mutated kinase plays a protective role in β-adrenergic receptor-stimulated cardiac myocyte apoptosis and myocardial remodelingCerrone R Foster, Mahipal Singh, Venkateswaran Subramanian, et al.Heart Failure Reviews|February 4, 2010
Role of osteopontin in heart failure associated with agingMahipal Singh, Cerrone R Foster, Suman Dalal, et al.Journal of Molecular and Cellular Cardiology|July 4, 2009
Osteopontin: role in extracellular matrix deposition and myocardial remodeling post-MIMahipal Singh, Cerrone R Foster, Suman Dalal, et al.Biochemical Society Transactions|February 4, 2020
The caveolar-mitochondrial interface: regulation of cellular metabolism in physiology and pathophysiologyCerrone R Foster, Shiho Satomi, Yuko Kato, et al.Experimental Physiology|December 20, 2011
Lack of ataxia telangiectasia mutated kinase induces structural and functional changes in the heart: role in β-adrenergic receptor-stimulated apoptosisCerrone R Foster, QinQin Zha, Laura L Daniel, et al.Molecular and Cellular Biochemistry|January 25, 2012
Β-adrenergic receptor stimulation induces endoplasmic reticulum stress in adult cardiac myocytes: role in apoptosisSuman Dalal, Cerrone R Foster, Bhudev C Das, et al.Journal of the American Heart Association|December 19, 2014
Deficiency of ataxia telangiectasia mutated kinase delays inflammatory response in the heart following myocardial infarctionLaura L Daniel, Christopher R Daniels, Saghar Harirforoosh, et al.Plos One|December 21, 2013
Deficiency of ataxia telangiectasia mutated kinase modulates cardiac remodeling following myocardial infarction: involvement in fibrosis and apoptosisCerrone R Foster, Laura L Daniel, Christopher R Daniels, et al.American Journal of Physiology. Heart and Circulatory Physiology|October 9, 2012
Exogenous ubiquitin modulates chronic β-adrenergic receptor-stimulated myocardial remodeling: role in Akt activity and matrix metalloproteinase expressionChristopher R Daniels, Cerrone R Foster, Sana Yakoob, et al.American Journal of Physiology. Heart and Circulatory Physiology|June 12, 2016
Ataxia telangiectasia-mutated kinase deficiency exacerbates left ventricular dysfunction and remodeling late after myocardial infarctionLaura L Daniel, Stephanie L C Scofield, Patsy Thrasher, et al.Pageof 1