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Scientific Reports|June 22, 2019
Myofibroblast modulation of cardiac myocyte structure and functionChandan K Nagaraju, Eef Dries, Guillaume Gilbert, et al.Cardiovascular Research|December 26, 2013
Reversible and irreversible differentiation of cardiac fibroblastsRonald B Driesen, Chandan K Nagaraju, Joëlle Abi-Char, et al.Scientific Reports|September 9, 2017
Global fibroblast activation throughout the left ventricle but localized fibrosis after myocardial infarctionChandan K Nagaraju, Eef Dries, Natasa Popovic, et al.Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|October 13, 2010
Glycogen synthase kinase-3α/β inhibition promotes in vivo amplification of endogenous mesenchymal progenitors with osteogenic and adipogenic potential and their differentiation to the osteogenic lineageAlessandra Gambardella, Chandan K Nagaraju, Patrick J O'Shea, et al.Communications Engineering|November 14, 2024
Precision sampling of discrete sites identified during in-vivo functional testing in the mammalian heartDylan Vermoortele, Camilla Olianti, Matthew Amoni, et al.American Journal of Physiology. Heart and Circulatory Physiology|September 11, 2016
Reduced mitochondrial respiration in the ischemic as well as in the remote nonischemic region in postmyocardial infarction remodelingDiogo T Galan, Virginie Bito, Piet Claus, et al.Cardiovascular Research|April 19, 2018
Hyperactive ryanodine receptors in human heart failure and ischaemic cardiomyopathy reside outside of couplonsEef Dries, Demetrio J Santiago, Guillaume Gilbert, et al.Journal of the American College of Cardiology|May 11, 2019
Myofibroblast Phenotype and Reversibility of Fibrosis in Patients With End-Stage Heart FailureChandan K Nagaraju, Emma L Robinson, Mouna Abdesselem, et al.The Journal of Physiology|January 5, 2020
Altered adrenergic response in myocytes bordering a chronic myocardial infarction underlies in vivo triggered activity and repolarization instabilityEef Dries, Matthew Amoni, Bert Vandenberk, et al.Basic Research in Cardiology|November 15, 2022
InsP<sub>3</sub>R-RyR Ca<sup>2+</sup> channel crosstalk facilitates arrhythmias in the failing human ventricleXin Jin, Matthew Amoni, Guillaume Gilbert, et al.Pageof 2