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Changli Wei

Showing results (51-60 of 79) with videos related to

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Science Translational Medicine|September 20, 2023
The D2D3 form of uPAR acts as an immunotoxin and may cause diabetes and kidney diseaseKe Zhu, Kamalika Mukherjee, Changli Wei, et al.
The Journal of Biological Chemistry|June 20, 2008
Neph-Nephrin proteins bind the Par3-Par6-atypical protein kinase C (aPKC) complex to regulate podocyte cell polarityBjörn Hartleben, Heiko Schweizer, Pauline Lübben, et al.
The New England Journal of Medicine|November 6, 2015
Soluble Urokinase Receptor and Chronic Kidney DiseaseSalim S Hayek, Sanja Sever, Yi-An Ko, et al.
The Journal of Biological Chemistry|May 13, 2014
Reduction of proteinuria through podocyte alkalinizationMehmet M Altintas, Kumiko Moriwaki, Changli Wei, et al.
Science Translational Medicine|October 3, 2014
A circulating antibody panel for pretransplant prediction of FSGS recurrence after kidney transplantationMarianne Delville, Tara K Sigdel, Changli Wei, et al.
Hybridoma (2005)|October 20, 2011
In vivo electroporation and non-protein based screening assays to identify antibodies against native protein conformationsPirouz Daftarian, Raquibul Chowdhury, Philip Ames, et al.
The Journal of Clinical Investigation|August 3, 2007
Proteolytic processing of dynamin by cytoplasmic cathepsin L is a mechanism for proteinuric kidney diseaseSanja Sever, Mehmet M Altintas, Sharif R Nankoe, et al.
Nature Communications|July 21, 2023
SuPAR mediates viral response proteinuria by rapidly changing podocyte functionChangli Wei, Prasun K Datta, Florian Siegerist, et al.
Kidney International|February 7, 2023
The small GTPase regulatory protein Rac1 drives podocyte injury independent of cationic channel protein TRPC5Onur K Polat, Elena Isaeva, Yashwanth R Sudhini, et al.
Science Signaling|September 9, 2011
Small molecule-mediated activation of the integrin CD11b/CD18 reduces inflammatory diseaseDony Maiguel, Mohd Hafeez Faridi, Changli Wei, et al.
Pageof 8

Showing results (51-60 of 79) with videos related to

Sort By:
Pageof 8
Science Translational Medicine|September 20, 2023
The D2D3 form of uPAR acts as an immunotoxin and may cause diabetes and kidney diseaseKe Zhu, Kamalika Mukherjee, Changli Wei, et al.
The Journal of Biological Chemistry|June 20, 2008
Neph-Nephrin proteins bind the Par3-Par6-atypical protein kinase C (aPKC) complex to regulate podocyte cell polarityBjörn Hartleben, Heiko Schweizer, Pauline Lübben, et al.
The New England Journal of Medicine|November 6, 2015
Soluble Urokinase Receptor and Chronic Kidney DiseaseSalim S Hayek, Sanja Sever, Yi-An Ko, et al.
The Journal of Biological Chemistry|May 13, 2014
Reduction of proteinuria through podocyte alkalinizationMehmet M Altintas, Kumiko Moriwaki, Changli Wei, et al.
Science Translational Medicine|October 3, 2014
A circulating antibody panel for pretransplant prediction of FSGS recurrence after kidney transplantationMarianne Delville, Tara K Sigdel, Changli Wei, et al.
Hybridoma (2005)|October 20, 2011
In vivo electroporation and non-protein based screening assays to identify antibodies against native protein conformationsPirouz Daftarian, Raquibul Chowdhury, Philip Ames, et al.
The Journal of Clinical Investigation|August 3, 2007
Proteolytic processing of dynamin by cytoplasmic cathepsin L is a mechanism for proteinuric kidney diseaseSanja Sever, Mehmet M Altintas, Sharif R Nankoe, et al.
Nature Communications|July 21, 2023
SuPAR mediates viral response proteinuria by rapidly changing podocyte functionChangli Wei, Prasun K Datta, Florian Siegerist, et al.
Kidney International|February 7, 2023
The small GTPase regulatory protein Rac1 drives podocyte injury independent of cationic channel protein TRPC5Onur K Polat, Elena Isaeva, Yashwanth R Sudhini, et al.
Science Signaling|September 9, 2011
Small molecule-mediated activation of the integrin CD11b/CD18 reduces inflammatory diseaseDony Maiguel, Mohd Hafeez Faridi, Changli Wei, et al.
Pageof 8