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Arthritis & Rheumatology (Hoboken, N.J.)|February 9, 2021
Effects of the TNFRSF11B Mutation Associated With Calcium Pyrophosphate Deposition Disease in Osteoclastogenesis in a Murine ModelElizabeth Mitton-Fitzgerald, Claudia M Gohr, Charlene J Williams, et al.
Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|May 8, 2009
Oxygen tension regulates the expression of ANK (progressive ankylosis) in an HIF-1-dependent manner in growth plate chondrocytesRaihana Zaka, Arnold S Dion, Anna Kusnierz, et al.
Journal of Cellular Biochemistry|April 16, 2005
Transforming growth factor-beta1 (TGF-beta1) regulates ATDC5 chondrogenic differentiation and fibronectin isoform expressionFei Han, Christopher S Adams, Zhuliang Tao, et al.
Arthritis and Rheumatism|September 18, 2003
Mutations in the amino terminus of ANKH in two US families with calcium pyrophosphate dihydrate crystal deposition diseaseCharlene J Williams, Adrian Pendleton, Gina Bonavita, et al.
The EMBO Journal|March 27, 2004
Regulatory regions and critical residues of NOD2 involved in muramyl dipeptide recognitionTsuyoshi Tanabe, Mathias Chamaillard, Yasunori Ogura, et al.
Plos Genetics|July 9, 2020
The membrane protein ANKH is crucial for bone mechanical performance by mediating cellular export of citrate and ATPFlora Szeri, Stefan Lundkvist, Sylvia Donnelly, et al.
American Journal of Human Genetics|September 26, 2002
Autosomal dominant familial calcium pyrophosphate dihydrate deposition disease is caused by mutation in the transmembrane protein ANKHCharlene J Williams, Yun Zhang, Andrew Timms, et al.
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