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Journal of Molecular and Cellular Cardiology|June 12, 2012
Embryonic expression of AMPK γ subunits and the identification of a novel γ2 transcript variant in adult heartKatalin Pinter, Robert T Grignani, Gabor Czibik, et al.
The Biochemical Journal|February 21, 2002
Two mutations in troponin I that cause hypertrophic cardiomyopathy have contrasting effects on cardiac muscle contractilityDavid Burton, Hassan Abdulrazzak, Adam Knott, et al.
The Journal of Biological Chemistry|August 21, 2002
Alterations in thin filament regulation induced by a human cardiac troponin T mutant that causes dilated cardiomyopathy are distinct from those induced by troponin T mutants that cause hypertrophic cardiomyopathyPaul Robinson, Mahmooda Mirza, Adam Knott, et al.
Circulation Research|October 19, 2002
Identification of novel interactions between domains of Myosin binding protein-C that are modulated by hypertrophic cardiomyopathy missense mutationsJohanna Moolman-Smook, Emily Flashman, Willem de Lange, et al.
The Journal of Biological Chemistry|May 16, 2018
Hypertrophic cardiomyopathy mutations increase myofilament Ca2+ buffering, alter intracellular Ca2+ handling, and stimulate Ca2+-dependent signalingPaul Robinson, Xing Liu, Alexander Sparrow, et al.
The Journal of Biological Chemistry|June 1, 2005
Dilated cardiomyopathy mutations in three thin filament regulatory proteins result in a common functional phenotypeMahmooda Mirza, Steven Marston, Ruth Willott, et al.
Journal of the American College of Cardiology|November 16, 2004
Severe disease expression of cardiac troponin C and T mutations in patients with idiopathic dilated cardiomyopathyJens Mogensen, Ross T Murphy, Tony Shaw, et al.
Circulation Research|November 17, 2007
Reduced phospholamban phosphorylation is associated with impaired relaxation in left ventricular myocytes from neuronal NO synthase-deficient miceYin Hua Zhang, Mei Hua Zhang, Claire E Sears, et al.
The Journal of Biological Chemistry|March 16, 2007
The effect of mutations in alpha-tropomyosin (E40K and E54K) that cause familial dilated cardiomyopathy on the regulatory mechanism of cardiac muscle thin filamentsMahmooda Mirza, Paul Robinson, Elena Kremneva, et al.
American Journal of Physiology. Heart and Circulatory Physiology|July 4, 2020
Dilated cardiomyopathy mutations in thin-filament regulatory proteins reduce contractility, suppress systolic Ca2+, and activate NFAT and Akt signalingPaul Robinson, Alexander J Sparrow, Suketu Patel, et al.
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